Novel TENM3–ALK fusion is an alternate mechanism for ALK activation in neuroblastoma - Hiwatari, Mitsuteru; Seki, Masafumi; Matsuno, Ryosuke; Yoshida, Kenichi; Nagasawa, Takeshi; Sato-Otsubo, Aiko; Yamamoto, Shohei; Kato, Motohiro; Watanabe, Kentaro; Sekiguchi, Masahiro; Miyano, Satoru; Ogawa, Seishi; et al. (Oncogene, 2022)
Keywords: 13/1; 13/106; 13/109; 13/44; 14/32; 38/109; 38/61; 38/77; 38/91; 631/67/2332; 631/67/395; 631/80/304; 64/60; 82/1; 82/80; AKT protein; Anaplastic Lymphoma Kinase - genetics; Animals; Apoptosis; Cell Biology; Cell Line, Tumor; Cell Proliferation; Cell Transformation, Neoplastic - genetics; Chromosome 19; Extracellular signal-regulated kinase; Fusion protein; Human Genetics; Humans; Internal Medicine; Kinases; Medicine; Medicine & Public Health; Membrane Proteins - genetics; Mice; Mice, Inbred NOD; Nerve Tissue Proteins - genetics; Neuroblastoma; Neuroblastoma - pathology; NIH 3T3 Cells; Oncogene Proteins, Fusion - genetics; Oncology; Patients; Protein-tyrosine kinase; Proteins; Receptor Protein-Tyrosine Kinases - metabolism; Stat3 protein; Therapeutic targets; Translocation, Genetic - genetics; Tumor cells; Tumorigenesis