Ketamine normalizes binge drinking-induced defects in glutamatergic synaptic transmission and ethanol drinking behavior in female but not male mice - Crowley, Nicole A.; Magee, Sarah N.; Feng, Mengyang; Jefferson, Sarah J.; Morris, Christian J.; Dao, Nigel C.; Brockway, Dakota F.; Luscher, Bernhard (Neuropharmacology, 2019)
Keywords: Alcohol; Animals; Binge Drinking - metabolism; Binge Drinking - therapy; Brain - drug effects; Brain - metabolism; Drinking in the dark; Elongation Factor 2 Kinase - metabolism; Excitatory Postsynaptic Potentials - drug effects; Female; Glutamic Acid - metabolism; Ketamine; Ketamine - pharmacology; Male; Mice; Mice, Inbred C57BL; Models, Animal; Prefrontal Cortex; Prelimbic cortex; Receptors, Glutamate - metabolism; Signal Transduction - drug effects; Synaptic Transmission - drug effects; Synaptic Transmission - physiology; TOR Serine-Threonine Kinases - metabolism; Alcohol; Prelimbic cortex; Ketamine; Drinking in the dark