ATF3/SPI1/SLC31A1 Signaling Promotes Cuproptosis Induced by Advanced Glycosylation End Products in Diabetic Myocardial Injury - Huo, Shengqi; Wang, Qian; Shi, Wei; Peng, Lulu; Jiang, Yue; Zhu, Mengying; Guo, Junyi; Peng, Dewei; Wang, Moran; Men, Lintong; Huang, Bingyu; Lv, Jiagao; et al. (International journal of molecular sciences, 2023)
Keywords: Animals; Apoptosis; Cardiac function; Cardiomyocytes; Cardiomyopathy; Cardiotoxicity - drug therapy; Cardiotoxicity - genetics; Cardiotoxicity - metabolism; Copper; Copper - metabolism; Cytotoxicity; Dehydrogenases; Diabetes; Diabetes Mellitus, Experimental - metabolism; Diabetic Cardiomyopathies - drug therapy; Diabetic Cardiomyopathies - genetics; Diabetic Cardiomyopathies - metabolism; Glycation End Products, Advanced - metabolism; Glycosylation; Homeostasis; Metabolic disorders; Mice; Myocytes, Cardiac - metabolism; Oxidative stress; Proteins; advanced glycosylation end products; diabetic cardiomyopathy; cuproptosis; SLC31A1