Glucagon-like Peptide 1 Receptor Signaling in Acinar Cells Causes Growth-Dependent Release of Pancreatic Enzymes - Wewer Albrechtsen, Nicolai J.; Albrechtsen, Reidar; Bremholm, Lasse; Svendsen, Berit; Kuhre, Rune E.; Poulsen, Steen S.; Christiansen, Charlotte B.; Jensen, Elisa P.; Janus, Charlotte; Hilsted, Linda; Deacon, Carolyn F.; Hartmann, Bolette; et al. (Cell reports (Cambridge), 2016)
Keywords: acinar cells; Acinar Cells - drug effects; Acinar Cells - enzymology; amylase; Amylases - genetics; Animals; Cell Line; Cell Proliferation - drug effects; Diabetes Mellitus, Type 2 - drug therapy; Diabetes Mellitus, Type 2 - enzymology; Forkhead Box Protein O1 - genetics; Gene Expression Regulation, Enzymologic; GLP-1; GLP-1R; Glucagon-Like Peptide 1 - administration & dosage; Glucagon-Like Peptide 1 - genetics; Glucagon-Like Peptide-1 Receptor - agonists; Glucagon-Like Peptide-1 Receptor - genetics; Humans; Incretins - therapeutic use; lipase; Lipase - genetics; Pancreas - enzymology; pancreatic enzymes; Pancreatitis - chemically induced; Pancreatitis - genetics; Pancreatitis - pathology; Signal Transduction; pancreatic enzymes; GLP-1; acinar cells; amylase; lipase; GLP-1R