Epigenetic silencing of IRF1 dysregulates type III interferon responses to respiratory virus infection in epithelial to mesenchymal transition - Yang, Jun; Tian, Bing; Sun, Hong; Garofalo, Roberto P.; Brasier, Allan R. (Nature microbiology, 2017)
Keywords: 14; 14/63; 38; 38/77; 42; 42/41; 631/250/262; 631/326/596/2555; 631/326/596/2557; 64; 64/60; 692/420/254; Allergic diseases; Antiviral Agents - metabolism; Cells, Cultured; Chronic infection; Epigenesis, Genetic; Epigenetics; Epithelial Cells - physiology; Epithelial Cells - virology; Epithelial-Mesenchymal Transition; Fibrosis; Gene Silencing; Homeobox; Host-Pathogen Interactions; Humans; Immune Evasion; Infectious Diseases; Interferon; Interferon regulatory factor; Interferon regulatory factor 1; Interferon regulatory factor 3; Interferon Regulatory Factor-1 - biosynthesis; Interferons - metabolism; Life Sciences; Medical Microbiology; Mesenchyme; Microbiology; Mucosa; Parasitology; Polycomb group proteins; Respiratory syncytial virus; Respiratory Syncytial Viruses - physiology; Respiratory tract diseases; Rhinovirus; Transforming growth factor-b; Virology; Zinc Finger E-box-Binding Homeobox 1 - metabolism; Zinc finger proteins