PKCγ promotes axonal remodeling in the cortico-spinal tract via GSK3β/β-catenin signaling after traumatic brain injury - Zhang, Bo; Li, Zaiwang; Zhang, Rui; Hu, Yaling; Jiang, Yingdi; Cao, Tingting; Wang, Jingjing; Gong, Lingli; Ji, Li; Mu, Huijun; Yang, Xusheng; Dai, Youai; et al. (Scientific reports, 2019)
Keywords: 13/1; 13/100; 13/109; 13/44; 13/51; 14/19; 14/34; 14/63; 631/378/1687; 631/378/340; 64/60; Animals; Axon guidance; Axonogenesis; Axons - drug effects; Axons - metabolism; beta Catenin - genetics; beta Catenin - metabolism; Brain Injuries, Traumatic - physiopathology; Fibers; Glycogen Synthase Kinase 3 beta - genetics; Glycogen Synthase Kinase 3 beta - metabolism; Humanities and Social Sciences; Male; Mice; Mice, Inbred BALB C; multidisciplinary; Nerve Regeneration; Neurons - cytology; Neurons - drug effects; Neurons - metabolism; Phosphorylation; Protein kinase C; Protein Kinase C - pharmacology; Pyramidal tracts; Pyramidal Tracts - cytology; Pyramidal Tracts - drug effects; Pyramidal Tracts - metabolism; Recovery of Function; Science; Science (multidisciplinary); Sensorimotor system; Signal Transduction; Spinal cord; Spinal cord injuries; Therapeutic applications; Traumatic brain injury; β-Catenin