Epithelial NSD2 maintains FMO‐mediated taurine biosynthesis to prevent intestinal barrier disruption - Xu, Yue; Xiao, Xiuying; Ma, Chunxiao; Wang, Ziyi; Feng, Wenxin; Rao, Hanyu; Zhang, Wei; Liu, Ningyuan; Aji, Rebiguli; Meng, Xiangjun; Gao, Wei‐Qiang; Li, Li (Clinical and translational medicine, 2024)
Keywords: Animals; Biosynthesis; Disease Models, Animal; histone methylation; Histone-Lysine N-Methyltransferase - genetics; Histone-Lysine N-Methyltransferase - metabolism; Humans; IBD; Inflammation; Inflammatory bowel disease; Inflammatory Bowel Diseases - genetics; Inflammatory Bowel Diseases - metabolism; Inflammatory Bowel Diseases - pathology; intestinal epithelial homeostasis; Intestinal Mucosa - metabolism; Mice; Mice, Knockout; NSD2; Taurine - metabolism; taurine biosynthesis; histone methylation; IBD; taurine biosynthesis; intestinal epithelial homeostasis; NSD2