MSCs ameliorate hepatocellular apoptosis mediated by PINK1-dependent mitophagy in liver ischemia/reperfusion injury through AMPKα activation - Zheng, Jun; Chen, Liang; Lu, Tongyu; Zhang, Yingcai; Sui, Xin; Li, Yang; Huang, Xuna; He, Liying; Cai, Jianye; Zhou, Chaorong; Liang, Jinliang; Chen, Guihua; et al. (Cell death & disease, 2020)
Keywords: 13/100; 13/109; 13/2; 13/21; 13/31; 13/51; 14/19; 14/28; 631/532/2074; 631/80/39/2348; 631/80/82/23; 82/80; 82/83; Animals; Antibodies; Apoptosis; Apoptosis - drug effects; Biochemistry; Biomedical and Life Sciences; Carcinoma, Hepatocellular - drug therapy; Carcinoma, Hepatocellular - metabolism; Cell Biology; Cell Culture; Culture Media, Conditioned - pharmacology; Hepatocytes; Hepatocytes - drug effects; Hepatocytes - metabolism; Hypoxia; Immunology; Ischemia; Ischemia - metabolism; Life Sciences; Liver; Liver Neoplasms - drug therapy; Liver Neoplasms - metabolism; Male; Mesenchymal Stem Cells - drug effects; Mesenchymal Stem Cells - metabolism; Mesenchyme; Mice, Inbred C57BL; Mitochondria; Mitochondria - drug effects; Mitochondria - metabolism; Mitophagy; Mitophagy - drug effects; Parkin protein; PTEN-induced putative kinase; Reactive oxygen species; Reactive Oxygen Species - metabolism; Reperfusion; Reperfusion Injury - drug therapy; Reperfusion Injury - metabolism; siRNA; Stem cell transplantation; Stem cells