The Axin/TNKS complex interacts with KIF3A and is required for insulin-stimulated GLUT4 translocation - Guo, Hui-Ling; Zhang, Cixiong; Liu, Qi; Li, Qinxi; Lian, Guili; Wu, Di; Li, Xuebin; Zhang, Wei; Shen, Yuemao; Ye, Zhiyun; Lin, Shu-Yong; Lin, Sheng-Cai (Cell research, 2012)
Keywords: 2型糖尿病; 3T3-L1 Cells; 631/80/2023/2022; Adipocytes - drug effects; Adipocytes - metabolism; Animals; Axi; Axin Protein - genetics; Axin Protein - metabolism; Biomedical and Life Sciences; Blood; Blood Glucose - analysis; Blood Glucose - drug effects; Blood Glucose - metabolism; Cell Biology; Cell Membrane - genetics; Cell Membrane - metabolism; Enzyme Activation; Glucose; Glucose Transporter Type 4 - genetics; Glucose Transporter Type 4 - metabolism; HEK293 Cells; Humans; Insulin; Insulin - administration & dosage; Insulin - pharmacology; Insulin Resistance; Kinesin - genetics; Kinesin - metabolism; Life Sciences; Male; Mice; Microscopy, Fluorescence; n蛋白; Original; original-article; Protein Interaction Mapping; Protein Stability; Protein Transport; RNA Interference; Signal Transduction; Tankyrases - genetics; Tankyrases - metabolism; trans-Golgi Network - metabolism; Translocation; Two-Hybrid System Techniques; 交互; 胰岛素; 蛋白复合物; 马达蛋白; 驱动蛋白; GLUT4 translocation; Axin; TNKS; KIF3A