Unphosphorylated STAT1 binds to the BST2 transcription promoter, promoting increased AKBA anchoring on HPMECs to alleviate ARDS - Li, Kaili; Zhou, Zixiang; Liu, Feng; Huang, Zuotian; Chen, Xiaoying; Zhou, Fachun (Scientific reports, 2025)
Keywords: 631/154; 631/80; 692/308; Acute respiratory distress syndrome; Animals; Antigens, CD - genetics; Antigens, CD - metabolism; Apoptosis - drug effects; Bone Marrow Stromal Antigen 2; Bone marrow stromal cell antigen 2; Cell Movement - drug effects; Cell Proliferation - drug effects; Endothelial Cells - drug effects; Endothelial Cells - metabolism; GPI-Linked Proteins - genetics; GPI-Linked Proteins - metabolism; Humanities and Social Sciences; Humans; Male; Mice; Molecular Docking Simulation; multidisciplinary; Phosphorylation; Promoter Regions, Genetic; Protein Binding; Proteomics; Respiratory Distress Syndrome - drug therapy; Respiratory Distress Syndrome - genetics; Respiratory Distress Syndrome - metabolism; Respiratory Distress Syndrome - pathology; Science; Science (multidisciplinary); Signal transducer and activator of transcription 1; STAT1 Transcription Factor - genetics; STAT1 Transcription Factor - metabolism; Transcriptomics; Acute respiratory distress syndrome; Signal transducer and activator of transcription 1; Bone marrow stromal cell antigen 2; Transcriptomics; Proteomics