CCL24 contributes to HCC malignancy via RhoB- VEGFA-VEGFR2 angiogenesis pathway and indicates poor prognosis - Jin, Lei; Liu, Wei-Ren; Tian, Meng-Xin; Jiang, Xi-Fei; Wang, Han; Zhou, Pei-Yun; Ding, Zhen-Bin; Peng, Yuan-Fei; Dai, Zhi; Qiu, Shuang-Jian; Zhou, Jian; Fan, Jia; et al. (Oncotarget, 2017)
Keywords: Animals; Carcinoma, Hepatocellular - genetics; Carcinoma, Hepatocellular - metabolism; Carcinoma, Hepatocellular - pathology; Cell Line, Tumor; Chemokine CCL24 - genetics; Chemokine CCL24 - metabolism; Female; Gene Expression Regulation, Neoplastic; Human Umbilical Vein Endothelial Cells; Humans; Liver Neoplasms - genetics; Liver Neoplasms - metabolism; Liver Neoplasms - pathology; Male; Mice; Neoplasm Transplantation; Prognosis; Receptor, Fibroblast Growth Factor, Type 2 - metabolism; Receptors, CCR3 - metabolism; Research Paper; rhoB GTP-Binding Protein - metabolism; Survival Analysis; Th2 Cells - metabolism; Up-Regulation; Vascular Endothelial Growth Factor A - metabolism; CCL24; HCC; prognosis; RhoB; VEGFA