miR-219 Cooperates with miR-338 in Myelination and Promotes Myelin Repair in the CNS - Wang, Haibo; Moyano, Ana Lis; Ma, Zhangyan; Deng, Yaqi; Lin, Yifeng; Zhao, Chuntao; Zhang, Liguo; Jiang, Minqing; He, Xuelian; Ma, Zhixing; Lu, Fanghui; Xin, Mei; et al. (Developmental cell, 2017)
Keywords: Animals; Cell Differentiation - drug effects; Cell Lineage - drug effects; Central Nervous System - drug effects; Central Nervous System - metabolism; Central Nervous System - pathology; Demyelinating Diseases - genetics; Demyelinating Diseases - pathology; demyelinating injury; Disease Models, Animal; Disease Progression; Encephalomyelitis, Autoimmune, Experimental - genetics; Encephalomyelitis, Autoimmune, Experimental - pathology; Etv5; experimental autoimmune encephalomyelitis; Gene Deletion; gene regulatory network; Lecithins - pharmacology; Lingo1; Membrane Proteins - metabolism; Mice, Inbred C57BL; Mice, Knockout; microRNAs; MicroRNAs - genetics; MicroRNAs - metabolism; miR-219; miR-338; Multiple Sclerosis - genetics; Multiple Sclerosis - pathology; Multiple Sclerosis - therapy; Myelin Sheath - drug effects; Myelin Sheath - metabolism; Myelin Sheath - pathology; myelination; Nerve Regeneration - drug effects; Nerve Regeneration - genetics; Nerve Tissue Proteins - metabolism; Oligodendroglia - drug effects; Oligodendroglia - metabolism; Optic Nerve - pathology; Optic Nerve - ultrastructure; Phenotype; remyelination; Spinal Cord - drug effects; Spinal Cord - metabolism; Spinal Cord - pathology; Wound Healing - drug effects; Wound Healing - genetics; microRNAs; experimental autoimmune encephalomyelitis; Lingo1; myelination; miR-338; gene regulatory network; miR-219; remyelination; demyelinating injury; Etv5