Altered Levels of Visinin-Like Protein 1 Correspond to Regional Neuronal Loss in Alzheimer Disease and Frontotemporal Lobar Degeneration - Kirkwood, Caitlin M.; MacDonald, Matthew L.; Schempf, Tadhg A.; Vatsavayi, Anil V.; Ikonomovic, Milos D.; Koppel, Jeremy L.; Ding, Ying; Sun, Mai; Kofler, Julia K.; Lopez, Oscar L.; Yates, Nathan A.; Sweet, Robert A. (Journal of neuropathology and experimental neurology, 2016)
Keywords: Age of Onset; Aged; Aged, 80 and over; alpha-Synuclein - biosynthesis; alpha-Synuclein - genetics; Alzheimer Disease - cerebrospinal fluid; Alzheimer Disease - genetics; Alzheimer Disease - pathology; Amino Acid Sequence; Amyloid beta-Peptides - biosynthesis; Amyloid beta-Peptides - genetics; Autopsy; Biomarkers - analysis; Cell Death; Entorhinal Cortex - pathology; Female; Frontotemporal Lobar Degeneration - cerebrospinal fluid; Frontotemporal Lobar Degeneration - genetics; Frontotemporal Lobar Degeneration - pathology; Gyrus Cinguli - pathology; Humans; Male; Middle Aged; Molecular Sequence Data; Neurocalcin - cerebrospinal fluid; Neurocalcin - genetics; Neurons - pathology; Original; tau Proteins - biosynthesis; tau Proteins - genetics; Frontotemporal lobar degeneration; Human postmortem brain tissue; Alzheimer disease; Visinin-like 1 protein (Vilip-1)