MiR-142-3p ameliorates high glucose-induced renal tubular epithelial cell injury by targeting BOD1 - Zhao, Ningmin; Luo, Qing; Lin, Ruijuan; Li, Qiaoyan; Ma, Peizhi (Clinical and experimental nephrology, 2021)
Keywords: Animals; Apoptosis; Apoptosis - drug effects; Apoptosis - genetics; Blood Urea Nitrogen; Cell Cycle Proteins - genetics; Cell Cycle Proteins - metabolism; Cell division; Cell injury; Cell Line; Cell Survival - drug effects; Cell viability; Creatinine - blood; Diabetes mellitus; Diabetes Mellitus, Experimental - chemically induced; Diabetic Nephropathies; Diabetic nephropathy; Down-Regulation - drug effects; Down-Regulation - genetics; Enzyme-linked immunosorbent assay; Epithelial cells; Epithelial Cells - metabolism; Epithelial Cells - pathology; Flow cytometry; Gene Expression; Glucose - administration & dosage; Glucose - pharmacology; Humans; Kidney Tubules; Kidneys; Male; Medicine; Medicine & Public Health; MicroRNAs - genetics; MicroRNAs - metabolism; miRNA; Nephrology; Nephropathy; Original Article; Oxidative stress; Oxidative Stress - drug effects; Oxidative Stress - genetics; Rats; RNA, Messenger - metabolism; Streptozocin; Urology; Diabetic nephropathy; miR-142-3p; Renal tubular epithelial cell injury; BOD1