Overexpression of GOLPH3 Promotes Proliferation and Tumorigenicity in Breast Cancer via Suppression of the FOXO1 Transcription Factor - Zeng, Zhaolei; Lin, Huanxin; Zhao, Xiaohui; Liu, Guanglin; Wang, Xi; Xu, Ruihua; Chen, Kun; Li, Jun; Song, Libing (Clinical cancer research, 2012)
Keywords: Animals; Antineoplastic agents; Biological and medical sciences; Blotting, Western; Breast Neoplasms - genetics; Breast Neoplasms - metabolism; Breast Neoplasms - pathology; Cell Cycle - genetics; Cell Line; Cell Line, Tumor; Cell Proliferation; Cell Transformation, Neoplastic - genetics; Cell Transformation, Neoplastic - metabolism; Cyclin-Dependent Kinase Inhibitor Proteins - genetics; Cyclin-Dependent Kinase Inhibitor Proteins - metabolism; Female; Forkhead Box Protein O1; Forkhead Transcription Factors - genetics; Forkhead Transcription Factors - metabolism; Gene Expression Regulation, Neoplastic; Gynecology. Andrology. Obstetrics; Humans; Immunohistochemistry; Kaplan-Meier Estimate; Ki-67 Antigen - metabolism; Mammary gland diseases; Medical sciences; Membrane Proteins - genetics; Membrane Proteins - metabolism; Mice; Mice, Inbred BALB C; Mice, Nude; Middle Aged; Pharmacology. Drug treatments; Reverse Transcriptase Polymerase Chain Reaction; RNA Interference; Transplantation, Heterologous; Tumors; Cell proliferation; Mammary gland diseases; Breast disease; Toxicity; Gene overexpression; Breast cancer; Tumorigenicity; Malignant tumor; Transcription factor FOXO1; Cancer