Calorie-induced ER stress suppresses uroguanylin satiety signaling in diet-induced obesity - Kim, G W; Lin, J E; Snook, A E; Aing, A S; Merlino, D J; Li, P; Waldman, S A (Nutrition & diabetes, 2016)
Keywords: 13/95; 14; 14/63; 38; 42; 631/1647/334/1874/345; 631/1647/767/1424; 631/443/319/1642/393; 631/45/776/198; 64; 64/110; 82; 96; Animals; Arcuate Nucleus of Hypothalamus - metabolism; Caloric Restriction; Clinical Nutrition; Diabetes; Diet; Endoplasmic Reticulum Stress - physiology; Energy Intake; Epidemiology; Fatty Liver - therapy; Gene Expression Regulation; Gene Silencing; Glucose Intolerance - therapy; Hormone Replacement Therapy; Internal Medicine; Intestinal Mucosa - metabolism; Medicine; Medicine & Public Health; Metabolic Diseases; Mice; Mice, Inbred C57BL; Mice, Obese; Mice, Transgenic; Natriuretic Peptides - blood; Natriuretic Peptides - metabolism; Obesity - physiopathology; Obesity - therapy; Original; original-article; Postprandial Period; Receptors, Enterotoxin; Receptors, Guanylate Cyclase-Coupled - genetics; Receptors, Guanylate Cyclase-Coupled - metabolism; Receptors, Peptide - genetics; Receptors, Peptide - metabolism; Satiation; Signal Transduction; Taurochenodeoxycholic Acid - pharmacology; Tunicamycin - pharmacology