ST2 blockade mitigates peritoneal fibrosis induced by TGF‐β and high glucose - Kim, Yong Chul; Kim, Kyu Hong; Lee, Sunhwa; Jo, Ji‐won; Park, Jae Yoon; Park, Mi‐seon; Tsogbadrakh, Bodokhsuren; Lee, Jung Pyo; Lee, Jae Wook; Kim, Dong Ki; Oh, Kook‐Hwan; Jang, In‐Jin; et al. (Journal of cellular and molecular medicine, 2019)
Keywords: Animal models; Animals; Biomarkers; Cell culture; Cells, Cultured; Chlorhexidine; Disease Models, Animal; Effluents; Epithelial-Mesenchymal Transition - drug effects; Epithelium - pathology; Female; Fibroblasts; Fibronectin; Fibrosis; Gene expression; Glucose - toxicity; Growth factors; Heart attacks; Hemodialysis; Humans; Inflammation; Interleukin-1 Receptor-Like 1 Protein - antagonists & inhibitors; Interleukin-1 Receptor-Like 1 Protein - metabolism; Male; Mice, Inbred C57BL; Mice, Knockout; Middle Aged; Original; Peritoneal Dialysis; peritoneal fibrosis; Peritoneal Fibrosis - pathology; Peritoneum; Peritoneum - pathology; Preservation; Proportional Hazards Models; Snail protein; soluble ST2; ST2 blockade; Survival Analysis; Transforming Growth Factor beta - toxicity; Transforming growth factor-b; Tumorigenicity; β-Galactosidase; peritoneal fibrosis; ST2 blockade; peritoneal dialysis; soluble ST2