Acute hydrogen sulfide poisoning in a dairy farmer
INTRODUCTION. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who developed acute respiratory arrest from hydrogen sulfide poisoning while performing work as a dairy farmer. The subject entered a poorly ventilat...
Saved in:
Published in | Clinical toxicology (Philadelphia, Pa.) Vol. 45; no. 4; pp. 420 - 423 |
---|---|
Main Authors | , , , |
Format | Journal Article |
Language | English |
Published |
Philadelphia, PA
Taylor & Francis
01.05.2007
|
Subjects | |
Online Access | Get full text |
Cover
Loading…
Abstract | INTRODUCTION. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who developed acute respiratory arrest from hydrogen sulfide poisoning while performing work as a dairy farmer.
The subject entered a poorly ventilated tank containing degrading eggs and, within seconds, collapsed. Coworkers were able to extract him within minutes but he was apneic. He was intubated by emergency medical services and subsequently managed with supportive care in the intensive care unit. Upon admission, a powerful rotten egg scent was noted and a hydrogen sulfide poisoning was suspected. Serum analysis for the presence of thiosulfate confirmed the diagnosis. Nitrite therapy was not administered as the subject arrived outside of the therapeutic window of effectiveness and showed evidence of excellent oxygenation. His examinations following arrival were consistent with an anoxic brain injury which slowly improved several months after the incident with intensive neuro-rehabilitation.
Hydrogen sulfide is a mitochondrial toxin and inhibits cytochrome-aa(3) and prevents cellular aerobic metabolism. Therapies for toxic exposures include removal from the contaminated environment, ventilation with 100% oxygen, and nitrite therapy if administered immediately after exposure. Hyperbaric oxygen (HBO) therapy has anecdotal support and remains controversial.
Hydrogen sulfide is a significant occupational health hazard. Education, personal protective equipment, and early treatment are important in improving outcomes. |
---|---|
AbstractList | INTRODUCTION. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who developed acute respiratory arrest from hydrogen sulfide poisoning while performing work as a dairy farmer.
The subject entered a poorly ventilated tank containing degrading eggs and, within seconds, collapsed. Coworkers were able to extract him within minutes but he was apneic. He was intubated by emergency medical services and subsequently managed with supportive care in the intensive care unit. Upon admission, a powerful rotten egg scent was noted and a hydrogen sulfide poisoning was suspected. Serum analysis for the presence of thiosulfate confirmed the diagnosis. Nitrite therapy was not administered as the subject arrived outside of the therapeutic window of effectiveness and showed evidence of excellent oxygenation. His examinations following arrival were consistent with an anoxic brain injury which slowly improved several months after the incident with intensive neuro-rehabilitation.
Hydrogen sulfide is a mitochondrial toxin and inhibits cytochrome-aa(3) and prevents cellular aerobic metabolism. Therapies for toxic exposures include removal from the contaminated environment, ventilation with 100% oxygen, and nitrite therapy if administered immediately after exposure. Hyperbaric oxygen (HBO) therapy has anecdotal support and remains controversial.
Hydrogen sulfide is a significant occupational health hazard. Education, personal protective equipment, and early treatment are important in improving outcomes. INTRODUCTION. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who developed acute respiratory arrest from hydrogen sulfide poisoning while performing work as a dairy farmer.UNLABELLEDINTRODUCTION. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who developed acute respiratory arrest from hydrogen sulfide poisoning while performing work as a dairy farmer.The subject entered a poorly ventilated tank containing degrading eggs and, within seconds, collapsed. Coworkers were able to extract him within minutes but he was apneic. He was intubated by emergency medical services and subsequently managed with supportive care in the intensive care unit. Upon admission, a powerful rotten egg scent was noted and a hydrogen sulfide poisoning was suspected. Serum analysis for the presence of thiosulfate confirmed the diagnosis. Nitrite therapy was not administered as the subject arrived outside of the therapeutic window of effectiveness and showed evidence of excellent oxygenation. His examinations following arrival were consistent with an anoxic brain injury which slowly improved several months after the incident with intensive neuro-rehabilitation.CASE REPORTThe subject entered a poorly ventilated tank containing degrading eggs and, within seconds, collapsed. Coworkers were able to extract him within minutes but he was apneic. He was intubated by emergency medical services and subsequently managed with supportive care in the intensive care unit. Upon admission, a powerful rotten egg scent was noted and a hydrogen sulfide poisoning was suspected. Serum analysis for the presence of thiosulfate confirmed the diagnosis. Nitrite therapy was not administered as the subject arrived outside of the therapeutic window of effectiveness and showed evidence of excellent oxygenation. His examinations following arrival were consistent with an anoxic brain injury which slowly improved several months after the incident with intensive neuro-rehabilitation.Hydrogen sulfide is a mitochondrial toxin and inhibits cytochrome-aa(3) and prevents cellular aerobic metabolism. Therapies for toxic exposures include removal from the contaminated environment, ventilation with 100% oxygen, and nitrite therapy if administered immediately after exposure. Hyperbaric oxygen (HBO) therapy has anecdotal support and remains controversial.DISCUSSIONHydrogen sulfide is a mitochondrial toxin and inhibits cytochrome-aa(3) and prevents cellular aerobic metabolism. Therapies for toxic exposures include removal from the contaminated environment, ventilation with 100% oxygen, and nitrite therapy if administered immediately after exposure. Hyperbaric oxygen (HBO) therapy has anecdotal support and remains controversial.Hydrogen sulfide is a significant occupational health hazard. Education, personal protective equipment, and early treatment are important in improving outcomes.CONCLUSIONHydrogen sulfide is a significant occupational health hazard. Education, personal protective equipment, and early treatment are important in improving outcomes. Introduction. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who developed acute respiratory arrest from hydrogen sulfide poisoning while performing work as a dairy farmer. Case report. The subject entered a poorly ventilated tank containing degrading eggs and, within seconds, collapsed. Coworkers were able to extract him within minutes but he was apneic. He was intubated by emergency medical services and subsequently managed with supportive care in the intensive care unit. Upon admission, a powerful rotten egg scent was noted and a hydrogen sulfide poisoning was suspected. Serum analysis for the presence of thiosulfate confirmed the diagnosis. Nitrite therapy was not administered as the subject arrived outside of the therapeutic window of effectiveness and showed evidence of excellent oxygenation. His examinations following arrival were consistent with an anoxic brain injury which slowly improved several months after the incident with intensive neuro-rehabilitation. Discussion. Hydrogen sulfide is a mitochondrial toxin and inhibits cytochrome-aa3 and prevents cellular aerobic metabolism. Therapies for toxic exposures include removal from the contaminated environment, ventilation with 100% oxygen, and nitrite therapy if administered immediately after exposure. Hyperbaric oxygen (HBO) therapy has anecdotal support and remains controversial. Conclusion. Hydrogen sulfide is a significant occupational health hazard. Education, personal protective equipment, and early treatment are important in improving outcomes. Introduction. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who developed acute respiratory arrest from hydrogen sulfide poisoning while performing work as a dairy farmer. Case report. The subject entered a poorly ventilated tank containing degrading eggs and, within seconds, collapsed. Coworkers were able to extract him within minutes but he was apneic. He was intubated by emergency medical services and subsequently managed with supportive care in the intensive care unit. Upon admission, a powerful rotten egg scent was noted and a hydrogen sulfide poisoning was suspected. Serum analysis for the presence of thiosulfate confirmed the diagnosis. Nitrite therapy was not administered as the subject arrived outside of the therapeutic window of effectiveness and showed evidence of excellent oxygenation. His examinations following arrival were consistent with an anoxic brain injury which slowly improved several months after the incident with intensive neuro-rehabilitation. Discussion. Hydrogen sulfide is a mitochondrial toxin and inhibits cytochrome-aa sub(3) and prevents cellular aerobic metabolism. Therapies for toxic exposures include removal from the contaminated environment, ventilation with 100% oxygen, and nitrite therapy if administered immediately after exposure. Hyperbaric oxygen (HBO) therapy has anecdotal support and remains controversial. Conclusion. Hydrogen sulfide is a significant occupational health hazard. Education, personal protective equipment, and early treatment are important in improving outcomes. |
Author | Musser, Jeffrey Gerasimon, Gregg Rinard, John Bennett, Steven |
Author_xml | – sequence: 1 givenname: Gregg surname: Gerasimon fullname: Gerasimon, Gregg – sequence: 2 givenname: Steven surname: Bennett fullname: Bennett, Steven – sequence: 3 givenname: Jeffrey surname: Musser fullname: Musser, Jeffrey – sequence: 4 givenname: John surname: Rinard fullname: Rinard, John |
BackLink | http://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=18821726$$DView record in Pascal Francis https://www.ncbi.nlm.nih.gov/pubmed/17486486$$D View this record in MEDLINE/PubMed |
BookMark | eNqF0ctKxDAUBuAgI85FH8CNdKO70Zzm2uUweIMBN7oOaZqOkTYdk3Yxb2_LVAVFhMAJ4fsPSc4cTXzjLULngK8BS3wDjHHCGeYYACQGfIRmw9kyY5BNxv0Apmge4xvGRNIMTtAUBJW8XzOUrkzX2uR1X4Rma30Su6p0hU12jYuNd36bOJ_opNAu7JNSh9qGU3Rc6iras7Eu0Mvd7fP6Ybl5un9crzZLQylplyQDBrkUlqSAc9YXkoMgtmD93ak1wnAtMlOmIse6hFQUhLFMlEIYbWWRkwW6OvTdhea9s7FVtYvGVpX2tumiEpjyTPaP-g-mWNCUcfYvhIxLzgTt4cUIu7y2hdoFV-uwV5__1oPLEehodFUG7Y2L307KFEQ6OHFwJjQxBlsq41rdusa3QbtKAVbDJNWvSfZJ-JH8av5n5gOa0Jt7 |
CitedBy_id | crossref_primary_10_1111_nyas_13559 crossref_primary_10_1039_C0AN00597E crossref_primary_10_3390_s24020407 crossref_primary_10_1051_ata_2010024 crossref_primary_10_1155_2019_8061823 crossref_primary_10_1002_bkcs_12611 crossref_primary_10_3109_15563650_2010_508044 crossref_primary_10_3109_15563650_2011_565419 crossref_primary_10_1051_ata_2012010 crossref_primary_10_1016_j_apsusc_2021_149026 crossref_primary_10_1016_j_saa_2020_118457 crossref_primary_10_1016_j_jsps_2018_02_004 crossref_primary_10_1007_s10895_014_1461_5 crossref_primary_10_1016_j_medleg_2015_01_002 crossref_primary_10_3109_15563650_2013_799676 crossref_primary_10_1016_j_annfar_2011_12_007 crossref_primary_10_1155_2015_295241 crossref_primary_10_1007_s13181_017_0650_4 crossref_primary_10_1093_jas_skab143 crossref_primary_10_1016_j_forsciint_2009_12_058 crossref_primary_10_1093_jat_bku114 crossref_primary_10_1016_j_ccc_2021_03_014 crossref_primary_10_1029_2019GH000213 crossref_primary_10_1080_10408444_2023_2168177 crossref_primary_10_1039_c1em10425j crossref_primary_10_1080_15376516_2022_2121192 crossref_primary_10_1111_1556_4029_12015 crossref_primary_10_1111_nyas_13419 |
Cites_doi | 10.1093/occmed/46.5.367 10.1021/ed035p347 10.1016/j.ajem.2005.05.001 10.1046/j.1442-2026.2001.00220.x 10.1002/ajim.4700280109 10.3109/10408448409029321 10.2486/indhealth.42.83 10.1080/15298668191420738 10.1016/0300-483X(83)90054-9 10.3109/01480547809016045 10.1016/0736-4679(85)90216-1 10.1016/0041-008X(66)90112-8 10.1016/0041-008X(86)90140-7 10.1016/0735-6757(95)90094-2 |
ContentType | Journal Article |
Copyright | 2007 INIST-CNRS |
Copyright_xml | – notice: 2007 INIST-CNRS |
DBID | AAYXX CITATION IQODW CGR CUY CVF ECM EIF NPM 7U7 C1K 7ST 7T2 SOI 7X8 |
DOI | 10.1080/15563650601118010 |
DatabaseName | CrossRef Pascal-Francis Medline MEDLINE MEDLINE (Ovid) MEDLINE MEDLINE PubMed Toxicology Abstracts Environmental Sciences and Pollution Management Environment Abstracts Health and Safety Science Abstracts (Full archive) Environment Abstracts MEDLINE - Academic |
DatabaseTitle | CrossRef MEDLINE Medline Complete MEDLINE with Full Text PubMed MEDLINE (Ovid) Toxicology Abstracts Environmental Sciences and Pollution Management Health & Safety Science Abstracts Environment Abstracts MEDLINE - Academic |
DatabaseTitleList | MEDLINE MEDLINE - Academic Health & Safety Science Abstracts Toxicology Abstracts |
Database_xml | – sequence: 1 dbid: NPM name: PubMed url: https://proxy.k.utb.cz/login?url=http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed sourceTypes: Index Database – sequence: 2 dbid: EIF name: MEDLINE url: https://proxy.k.utb.cz/login?url=https://www.webofscience.com/wos/medline/basic-search sourceTypes: Index Database |
DeliveryMethod | fulltext_linktorsrc |
Discipline | Public Health |
EISSN | 1556-9519 |
EndPage | 423 |
ExternalDocumentID | 17486486 18821726 10_1080_15563650601118010 |
Genre | Journal Article Case Reports |
GroupedDBID | --- .GJ 00X 03L 0R~ 29B 34G 36B 39C 3O- 4.4 53G 5RE 5VS AAGDL AALIY AALUX AAMIU AAPUL AAQRR AAYXX ABBKH ABCRQ ABDBF ABEIZ ABJNI ABLIJ ABLKL ABUPF ABWVI ABXYU ACENM ACGFS ACIEZ ACKYO ACUHS ADCVX ADRBQ ADYSH AECIN AENEX AEOZL AFKVX AFRVT AGAFX AGDLA AGFJD AGRBW AGYJP AIJEM AIRBT AJEBJ AJWEG AKBVH ALMA_UNASSIGNED_HOLDINGS ALQZU ALYBC AMPGV AWYRJ BABNJ BLEHA BOHLJ CAG CCCUG CITATION COF DEIEU DKSSO DTRLO DU5 DZHFC EAP EAS EBC EBD EBS EDH EHN EJD EMB EMK EMOBN EPL EPT ESX F5P H13 HZ~ KRBQP KSSTO KWAYT KYCEM LJTGL LSO M44 M4Z NUSFT O9- QRXOQ Q~Q RNANH RVRKI SV3 TBQAZ TDBHL TERGH TFDNU TFL TFW TUROJ TUS UHWXJ V1S ~1N IQODW TASJS 0BK CGR CUY CVF ECM EIF NPM 7U7 C1K 7ST 7T2 SOI 7X8 |
ID | FETCH-LOGICAL-c443t-39151b87e3210b5e323b173ed51084ec7c6a79cf27b0af127d35597f77cae8db3 |
ISSN | 1556-3650 |
IngestDate | Thu Aug 07 14:24:38 EDT 2025 Fri Jul 11 03:23:33 EDT 2025 Thu Aug 07 14:30:44 EDT 2025 Wed Feb 19 02:07:22 EST 2025 Mon Jul 21 09:13:14 EDT 2025 Tue Jul 01 01:35:42 EDT 2025 Thu Apr 24 22:57:06 EDT 2025 |
IsPeerReviewed | true |
IsScholarly | true |
Issue | 4 |
Keywords | Human Toxic gas Cytochrome-aa3 Enzyme Acute Nitrites Cytochrome-c oxidase Hyperbaric oxygen Nitrite Case study Hydrogen sulfide Occupational accident Hyperbaric oxygen (HBO) Treatment Poisoning Oxidoreductases Farmer Occupational medicine |
Language | English |
License | CC BY 4.0 |
LinkModel | OpenURL |
MergedId | FETCHMERGED-LOGICAL-c443t-39151b87e3210b5e323b173ed51084ec7c6a79cf27b0af127d35597f77cae8db3 |
Notes | ObjectType-Article-1 SourceType-Scholarly Journals-1 ObjectType-Feature-2 content type line 23 ObjectType-Case Study-2 ObjectType-Feature-4 ObjectType-Report-1 ObjectType-Article-3 |
PMID | 17486486 |
PQID | 19686574 |
PQPubID | 23462 |
PageCount | 4 |
ParticipantIDs | proquest_miscellaneous_70469800 proquest_miscellaneous_20742565 proquest_miscellaneous_19686574 pubmed_primary_17486486 pascalfrancis_primary_18821726 crossref_citationtrail_10_1080_15563650601118010 crossref_primary_10_1080_15563650601118010 |
ProviderPackageCode | CITATION AAYXX |
PublicationCentury | 2000 |
PublicationDate | 2007-05-00 |
PublicationDateYYYYMMDD | 2007-05-01 |
PublicationDate_xml | – month: 05 year: 2007 text: 2007-05-00 |
PublicationDecade | 2000 |
PublicationPlace | Philadelphia, PA |
PublicationPlace_xml | – name: Philadelphia, PA – name: England |
PublicationTitle | Clinical toxicology (Philadelphia, Pa.) |
PublicationTitleAlternate | Clin Toxicol (Phila) |
PublicationYear | 2007 |
Publisher | Taylor & Francis |
Publisher_xml | – name: Taylor & Francis |
References | Prior M (CIT0007) 1988; 52 CIT0010 CIT0021 CIT0020 CIT0012 CIT0023 CIT0011 Marcus S (CIT0018) 1983; 5 Voight G (CIT0002) 1955; 1 Warenycia M (CIT0015) 1989; 38 Milby T (CIT0008) 1962; 4 CIT0003 Burnett W (CIT0014) 1977; 117 CIT0025 CIT0013 CIT0024 CIT0005 CIT0016 CIT0027 CIT0004 CIT0006 CIT0017 CIT0028 CIT0019 |
References_xml | – ident: CIT0023 doi: 10.1093/occmed/46.5.367 – volume: 1 start-page: 223 year: 1955 ident: CIT0002 publication-title: Acta Histochem – ident: CIT0005 doi: 10.1021/ed035p347 – ident: CIT0004 doi: 10.1016/j.ajem.2005.05.001 – ident: CIT0024 doi: 10.1046/j.1442-2026.2001.00220.x – volume: 117 start-page: 1277 year: 1977 ident: CIT0014 publication-title: Can Med Assoc J – ident: CIT0010 doi: 10.1093/occmed/46.5.367 – volume: 5 start-page: 1 year: 1983 ident: CIT0018 publication-title: Clin Toxicol Rev – volume: 52 start-page: 375 year: 1988 ident: CIT0007 publication-title: Can J Vet Res – ident: CIT0011 doi: 10.1002/ajim.4700280109 – ident: CIT0012 doi: 10.3109/10408448409029321 – ident: CIT0003 doi: 10.2486/indhealth.42.83 – ident: CIT0016 doi: 10.1080/15298668191420738 – ident: CIT0006 doi: 10.1016/0300-483X(83)90054-9 – volume: 38 start-page: 973 year: 1989 ident: CIT0015 publication-title: Demonstration of selective uptake of sulfide by the brainstem by measurement of brain sulfide levels. Biochem Pharmacol – ident: CIT0017 doi: 10.3109/01480547809016045 – ident: CIT0019 doi: 10.1016/0736-4679(85)90216-1 – ident: CIT0020 doi: 10.1016/0041-008X(66)90112-8 – ident: CIT0028 doi: 10.1016/0041-008X(86)90140-7 – volume: 4 start-page: 431 year: 1962 ident: CIT0008 publication-title: J Occup Med – ident: CIT0025 doi: 10.1093/occmed/46.5.367 – ident: CIT0013 doi: 10.1016/0735-6757(95)90094-2 – ident: CIT0021 doi: 10.1080/15298668191420738 – ident: CIT0027 doi: 10.1016/0736-4679(85)90216-1 |
SSID | ssj0038491 |
Score | 1.992366 |
Snippet | INTRODUCTION. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who... Introduction. Hydrogen sulfide is a lipid-soluble gas produced in occupational settings and from decaying organic matter. We present a 36-year-old man who... |
SourceID | proquest pubmed pascalfrancis crossref |
SourceType | Aggregation Database Index Database Enrichment Source |
StartPage | 420 |
SubjectTerms | Acute Disease Adult Agricultural Workers' Diseases - blood Agricultural Workers' Diseases - chemically induced Agricultural Workers' Diseases - rehabilitation Apnea - chemically induced Biological and medical sciences Chemical and industrial products toxicology. Toxic occupational diseases Dairying Eggs Gas, fumes Humans Hydrogen Sulfide - blood Hydrogen Sulfide - poisoning Hypoxia, Brain - chemically induced Hypoxia, Brain - physiopathology Hypoxia, Brain - rehabilitation Male Medical sciences Nervous System Diseases - blood Nervous System Diseases - chemically induced Nervous System Diseases - rehabilitation Occupational Exposure - adverse effects Occupational Exposure - analysis Thiosulfates - blood Toxicology |
Title | Acute hydrogen sulfide poisoning in a dairy farmer |
URI | https://www.ncbi.nlm.nih.gov/pubmed/17486486 https://www.proquest.com/docview/19686574 https://www.proquest.com/docview/20742565 https://www.proquest.com/docview/70469800 |
Volume | 45 |
hasFullText | 1 |
inHoldings | 1 |
isFullTextHit | |
isPrint | |
link | http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwnV1Lj9MwELZg94K0Qrwpj8UHTlQpeTh2cuzClhXaXTi0Um-R7Ti7lSCp2hSx_HrGsZ2kKuUlVWlkOXbkmYxn7Pk-I_Q6AB3giS88-NCkR3gM35xQqRcLmDwkg1gu0Xjni0t6NiMf5_G8oydo0CW1GMkfv8SV_I9UoQzkqlGy_yDZtlEogHuQL1xBwnD9KxmPpd7lv77JVxXU0ZnmxSJXw2WlU4QsWIUPc75Y3QwLvvpqM3EdMYEDRdbV94U0XEzgb-olFs0dubw2ebSf-ai3XPBBrfga5Fs6gMtVG9A3CJ_apo596yBmFxu95d-DjbWbPBoLnG8lBLv1B9Zl-zmTGVMvooY-dqS6MvDd0r6dNbSRVp9Iz2iS0O_Nv8Tgj3dMu8mF1C3rziCO1OR1NiV2i0b78lM2mZ2fZ9PT-fQ2OgwhfgADeDg-eX8ycZN0lJDUUOnal3cb3on_dqeLLZflaMnXIJrCHHuyPy5p_JPpPXTXBhZ4bLTkPrqlygfoyKzKYgM2e4jCRmOw0xhsNQa3GoMXJea40RhsNOYRmk1Op-_OPHtqhicJiWpPM_4HImFKo7NEDH-RCFikcrC-CVGSScpZKouQCZ8XQcjySEeVBWOSqyQX0WN0UFaleopwGsqUSRlwShnJqRIiDArp50oFPuOcD5DvBiaTllJen2zyJQss8-zOWA7Qm_aRpeFT-V3l463R7p6AuBA8bzpAr9zwZ2AW9V4XL1W1WWcwsSQ0ZmR_jVCvCkE4s78Ga45X9eE1nhjJdv0zklD4Pftj68_Rne6jeYEO6tVGvQQ3thbHViV_Allol8o |
linkProvider | EBSCOhost |
openUrl | ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Acute+hydrogen+sulfide+poisoning+in+a+dairy+farmer&rft.jtitle=Clinical+toxicology+%28Philadelphia%2C+Pa.%29&rft.au=Gerasimon%2C+Gregg&rft.au=Bennett%2C+Steven&rft.au=Musser%2C+Jeffrey&rft.au=Rinard%2C+John&rft.date=2007-05-01&rft.issn=1556-3650&rft.eissn=1556-9519&rft.volume=45&rft.issue=4&rft.spage=420&rft.epage=423&rft_id=info:doi/10.1080%2F15563650601118010&rft.externalDBID=NO_FULL_TEXT |
thumbnail_l | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=1556-3650&client=summon |
thumbnail_m | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=1556-3650&client=summon |
thumbnail_s | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=1556-3650&client=summon |