Complement factor H attenuates TNF-α-induced inflammation by upregulating EIF3C in rheumatoid arthritis - Jia, Yimeng; Feng, Bin; Ji, Xin; Tian, Xinping; Zhao, Lidan; Zhou, Jiaxin; Zhang, Wen; Li, Mengtao; Fei, Yunyun; Wu, Xunyao (Journal of translational medicine, 2023)
Keywords: Arthritis, Rheumatoid - drug therapy; C-reactive protein; Caspase-3; Cell death; Cell Proliferation; Cells; Cells, Cultured; Complement factor H; Complement Factor H - genetics; Complement Factor H - metabolism; Complement Factor H - therapeutic use; Correlation analysis; Cytokines; Cytokines - metabolism; Enzyme-linked immunosorbent assay; Enzymes; Erythrocyte sedimentation rate; Eukaryotic translation initiation factor 3 subunit C; Females; Fibroblast-like synoviocytes; Fibroblasts; Fibroblasts - metabolism; Flow cytometry; Humans; Inflammation; Inflammation - metabolism; Interleukin 6; Interleukin 8; Interleukin-6 - metabolism; Interleukin-8 - metabolism; Joint diseases; Leukocyte migration; Matrix metalloproteinase; Monocytes; Penicillin; Pyroptosis; Reagents; Rheumatoid arthritis; Software; Statistical analysis; Stromelysin 1; Synovial fluid; Synovial Membrane - metabolism; Synoviocytes; Therapeutic targets; Tumor Necrosis Factor-alpha - metabolism; Tumor necrosis factor-α; Western blotting; Wound healing; Beijing China; United States--US; China; Complement factor H; Fibroblast-like synoviocytes; Pyroptosis; Rheumatoid arthritis; Eukaryotic translation initiation factor 3 subunit C