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The relations of major platelet receptor expression during myocardial infarction. Monitoring efficacy of GPIIb/IIIa inhibitors by measuring P-selectin?

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Bibliographic Details
Published inThrombosis and haemostasis Vol. 81; no. 2; p. 314
Main Authors Serebruany, V L, Gurbel, P A
Format Journal Article
LanguageEnglish
Published Germany 01.02.1999
Subjects
Drug Evaluation
Fibrinolytic Agents - therapeutic use
Gene Expression Regulation
Humans
Myocardial Infarction - blood
Myocardial Infarction - drug therapy
Myocardial Infarction - genetics
Myocardial Reperfusion Injury - blood
Myocardial Reperfusion Injury - genetics
P-Selectin - biosynthesis
P-Selectin - genetics
Plasminogen Activators - therapeutic use
Platelet Endothelial Cell Adhesion Molecule-1 - biosynthesis
Platelet Endothelial Cell Adhesion Molecule-1 - genetics
Platelet Glycoprotein GPIIb-IIIa Complex - antagonists & inhibitors
Platelet Glycoprotein GPIIb-IIIa Complex - biosynthesis
Platelet Glycoprotein GPIIb-IIIa Complex - genetics
Receptors, Vitronectin - biosynthesis
Receptors, Vitronectin - genetics
Recombinant Proteins - therapeutic use
Thrombolytic Therapy
Tissue Plasminogen Activator - therapeutic use
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  • ikona citování Cite this
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