The relations of major platelet receptor expression during myocardial infarction. Monitoring efficacy of GPIIb/IIIa inhibitors by measuring P-selectin? - Serebruany, V L; Gurbel, P A (Thrombosis and haemostasis, 1999)
Keywords: Drug Evaluation; Fibrinolytic Agents - therapeutic use; Gene Expression Regulation; Humans; Myocardial Infarction - blood; Myocardial Infarction - drug therapy; Myocardial Infarction - genetics; Myocardial Reperfusion Injury - blood; Myocardial Reperfusion Injury - genetics; P-Selectin - biosynthesis; P-Selectin - genetics; Plasminogen Activators - therapeutic use; Platelet Endothelial Cell Adhesion Molecule-1 - biosynthesis; Platelet Endothelial Cell Adhesion Molecule-1 - genetics; Platelet Glycoprotein GPIIb-IIIa Complex - antagonists & inhibitors; Platelet Glycoprotein GPIIb-IIIa Complex - biosynthesis; Platelet Glycoprotein GPIIb-IIIa Complex - genetics; Receptors, Vitronectin - biosynthesis; Receptors, Vitronectin - genetics; Recombinant Proteins - therapeutic use; Thrombolytic Therapy; Tissue Plasminogen Activator - therapeutic use