p21 protects cardiomyocytes against ischemia-reperfusion injury by inhibiting oxidative stress - Li, Hong; Zou, Tong; Meng, Shuai; Peng, Yun‑Zhu; Yang, Jie‑Fu (Molecular medicine reports, 2018)
Keywords: Animals; Antioxidants; Biotechnology; Cardiomyocytes; Care and treatment; Cell cycle; Cell death; Cell Hypoxia; Cell Line; Cell Survival; Cellular signal transduction; Coronary artery disease; Cyclin-dependent kinase inhibitor p21; Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors; Cyclin-Dependent Kinase Inhibitor p21 - genetics; Cyclin-Dependent Kinase Inhibitor p21 - metabolism; Enzyme inhibitors; Enzymes; Fluorescent Dyes - chemistry; Gene expression; Glucose; GTP-binding protein; Health aspects; Heart diseases; Immunoglobulins; Ischemia; Kinases; Myocardial Reperfusion Injury - metabolism; Myocardial Reperfusion Injury - pathology; Myocytes; Myocytes, Cardiac - cytology; Myocytes, Cardiac - metabolism; NF-E2-Related Factor 2 - antagonists & inhibitors; NF-E2-Related Factor 2 - genetics; NF-E2-Related Factor 2 - metabolism; Oxidative Stress; Oxygen - metabolism; Oxygen consumption (Metabolism); p53 Protein; Polymerase chain reaction; Proteins; Rats; Reactive oxygen species; Reactive Oxygen Species - analysis; Reactive Oxygen Species - chemistry; Reperfusion; Reperfusion injury; Reverse transcription; RNA Interference; RNA, Small Interfering - metabolism; Rodents; Signal Transduction; Testing; Transcription factors; Tumor Suppressor Protein p53 - metabolism; Up-Regulation - drug effects; Western blotting; United States--US; China; Germany