Angiotensin-(1-7) attenuates damage to podocytes induced by preeclamptic serum through MAPK pathways - TIAN, JIMEI; ZHANG, LIHONG; ZHOU, YUNJIAO; XIAO, JING; LI, SHERAN; CHEN, YAPING; QIAO, ZHONGDONG; NIU, JIANYING; GU, YONG (International journal of molecular medicine, 2014)
Keywords: Abnormalities; Actins - metabolism; Adult; Age; Analysis; Angiotensin; Angiotensin I - pharmacology; Angiotensin II - analogs & derivatives; Angiotensin II - pharmacology; angiotensin-(1-7); Apoptosis; Apoptosis - drug effects; Birth weight; Cell Survival - drug effects; Diabetes; Drug dosages; Enzyme activation; Epithelium; Female; Genetic aspects; Humans; Hypertension; Intracellular Signaling Peptides and Proteins - metabolism; Kidney diseases; Kinases; MAP Kinase Signaling System - drug effects; Membrane Proteins - metabolism; mitogen-activated protein kinase phosphorylation; Mitogen-Activated Protein Kinases - metabolism; Pathogenesis; Peptide Fragments - pharmacology; Phosphorylation; Phosphorylation - drug effects; podocyte; Podocytes - drug effects; Podocytes - enzymology; Podocytes - pathology; Pre-Eclampsia - blood; Pre-Eclampsia - enzymology; Preeclampsia; Pregnancy; Properties; Proteins; Proto-Oncogene Proteins - metabolism; Receptors, G-Protein-Coupled - metabolism; Rodents; Studies; Uric acid; Urine; Womens health