Failure of nitric oxide donors to alter arrhythmias induced by acute myocardial ischaemia or reperfusion in anaesthetized rats - Barnes, Christopher S.; Coker, Susan J. (British journal of pharmacology, 1995)
Keywords: Anesthesia; Animals; Anti-Arrhythmia Agents - pharmacology; Antiarythmic agents; arrhythmias; Arrhythmias, Cardiac - etiology; Arrhythmias, Cardiac - physiopathology; Biological and medical sciences; Cardiovascular system; coronary artery occlusion; cyclic AMP; Cyclic AMP - metabolism; cyclic GMP; Cyclic GMP - metabolism; Hemodynamics - drug effects; Isosorbide Dinitrate - pharmacology; Male; Medical sciences; Molsidomine - analogs & derivatives; Molsidomine - pharmacology; myocardial ischaemia; Myocardial Ischemia - complications; Myocardial Ischemia - physiopathology; Myocardial Reperfusion Injury - complications; Myocardial Reperfusion Injury - physiopathology; Myocardium - metabolism; Nitric oxide; Nitric Oxide - physiology; Nitroprusside - pharmacology; Pharmacology. Drug treatments; Rats; Rats, Wistar; reperfusion; SIN‐1; sodium nitroprusside; Vasodilator Agents - pharmacology; Nitro compound; Vasodilator agent; Rat; Arrhythmia; Rodentia; Cardiovascular disease; Coronary heart disease; Myocardial disease; Vascular disease; Vertebrata; Chemotherapy; Experimental disease; Mammalia; Reperfusion; Treatment; Ischemia; Animal; Nitrogen monoxide; Myocardium; Cyclic nucleotide; Antiarrhythmia agent