IL-10/TGF-β–Modified Macrophages Induce Regulatory T Cells and Protect against Adriamycin Nephrosis - Cao, Qi; Wang, Yiping; Zheng, Dong; Sun, Yan; Wang, Ya; Lee, Vincent W.S.; Zheng, Guoping; Tan, Thian Kui; Ince, Jon; Alexander, Stephen I.; Harris, David C.H. (Journal of the American Society of Nephrology, 2010)
Keywords: Animals; B7-1 Antigen - metabolism; Basic Research; Biological and medical sciences; CD4-Positive T-Lymphocytes - drug effects; CD4-Positive T-Lymphocytes - metabolism; CD4-Positive T-Lymphocytes - pathology; Cell Proliferation - drug effects; Cells, Cultured; Disease Models, Animal; Doxorubicin - adverse effects; Glomerulonephritis; Interleukin-10 - pharmacology; Kidney - drug effects; Kidney - metabolism; Kidney - pathology; Macrophages - drug effects; Macrophages - pathology; Male; Medical sciences; Mice; Mice, Inbred BALB C; Nephrology. Urinary tract diseases; Nephropathies. Renovascular diseases. Renal failure; Nephrosis - chemically induced; Nephrosis - physiopathology; Nephrosis - prevention & control; Nitric Oxide Synthase Type II - metabolism; Phenotype; T-Lymphocytes, Regulatory - pathology; Transforming Growth Factor beta - pharmacology; V-Set Domain-Containing T-Cell Activation Inhibitor 1; Glomerulonephritis; Kidney disease; Antineoplastic agent; DNA topoisomerase (ATP-hydrolysing); Nephrology; Urinary system disease; Enzyme; Transforming growth factor β; Cytokine; Enzyme inhibitor; Nephrotic syndrome; Topoisomerase II inhibitor; Doxorubicin; Urology; Antibiotic; Isomerases; Interleukin 10; T-Lymphocyte; Anthracyclins; Regulatory cell; Macrophage