Receptor tyrosine kinase fusions act as a significant alternative driver of the serrated pathway in colorectal cancer development - Chan, Anthony W‐H; Pan, Yi; Tong, Joanna H‐M; Lung, Raymond W‐M; Kwan, Johnny S‐H; Chow, Chit; Tin, Edith K‐Y; Chung, Lau‐Ying; Li, Hui; Wong, Shela S‐Y; Chau, Shuk‐Ling; Chan, Yuk Yu; et al. (The Journal of pathology, 2020)
Keywords: Cell fusion; Cell proliferation; Colorectal cancer; Colorectal carcinoma; Ectopic expression; fusion genes; Fusion protein; Genetic transformation; Kinases; Lesions; MAP kinase; p53 Protein; Polyps; Protein-tyrosine kinase receptors; receptor tyrosine kinase; Ret protein; serrated pathway; sessile serrated lesion; Translocation; Tumorigenesis; Tumors; fusion genes; sessile serrated lesion; receptor tyrosine kinase; colorectal cancer; serrated pathway