RNA N6‐methyladenosine methyltransferase‐like 3 promotes liver cancer progression through YTHDF2‐dependent posttranscriptional silencing of SOCS2 - Chen, Mengnuo; Wei, Lai; Law, Cheuk‐Ting; Tsang, Felice Ho‐Ching; Shen, Jialing; Cheng, Carol Lai‐Hung; Tsang, Long‐Hin; Ho, Daniel Wai‐Hung; Chiu, David Kung‐Chun; Lee, Joyce Man‐Fong; Wong, Carmen Chak‐Lui; Ng, Irene Oi‐Lin; et al. (Hepatology (Baltimore, Md.), 2018)
Keywords: Adenosine; Animals; Carcinogenesis; Carcinoma, Hepatocellular - enzymology; Carcinoma, Hepatocellular - etiology; Cell migration; Cell proliferation; Chemical modification; Chromatin remodeling; CRISPR; Disease Progression; DNA methylation; Epigenetics; Gene expression; Hepatocellular carcinoma; Hepatology; Humans; Liver cancer; Liver Neoplasms - enzymology; Liver Neoplasms - etiology; Metastases; Methyltransferases - physiology; Mice; mRNA stability; N6-methyladenosine; Polymerase chain reaction; Post-transcription; RNA Interference; RNA modification; RNA-Binding Proteins - physiology; Solid tumors; Splicing; Suppressor of Cytokine Signaling Proteins - genetics; Tumorigenicity