Factors associated with severe effects following acute glufosinate poisoning

• Published in: Clinical toxicology (Philadelphia, Pa.) Vol. 51; no. 9; pp. 846 - 849
• Main Authors: Inoue, Y., Onodera, M., Fujita, Y., Fujino, Y., Kikuchi, S., Endo, S.
• Format: Journal Article
• Language: English
• Published: England Informa Healthcare 01.11.2013; Taylor & Francis
• Subjects: Adult; Aged; Aged, 80 and over; Aminobutyrates - toxicity; CNS/Psychological< Organ/tissue specific< Complications of poisoning; convulsion; Enzyme Inhibitors - toxicity; Female; FiO; Glutamate Decarboxylase - antagonists & inhibitors; Glutamate-Ammonia Ligase - antagonists & inhibitors; Herbicides - toxicity; Humans; Lung< Organ/tissue specific< Complications of poisoning; Male; Middle Aged; Neurotoxicity Syndromes - blood; Neurotoxicity Syndromes - immunology; Neurotoxicity Syndromes - physiopathology; Other; Oxygen - blood; PaO; Pulmonary Circulation - drug effects; ratio (P/F ratio); Respiratory arrest; Respiratory Insufficiency - etiology; Retrospective Studies; ROC Curve; Seizures - etiology; Severity of Illness Index; Shock index; Systemic inflammatory response syndrome; Systemic Inflammatory Response Syndrome - etiology; Young Adult
• ISSN: 1556-3650; 1556-9519; 1556-9519
• DOI: 10.3109/15563650.2013.841180

Abstract Context. In acute glufosinate poisoning, sudden respiratory arrest and convulsion can occur after a latent period of 4-60 h. There is still no factor that accurately predicts the occurrence of these symptoms. Objective. To elucidate the predictors of severe effects following acute glufosina...