Diminished NO generation by injured endothelium and loss of macula densa nNOS may contribute to sustained acute kidney injury after ischemia-reperfusion - Kwon, Osun; Hong, Seok-Min; Ramesh, Ganesan (American journal of physiology. Renal physiology, 2009)
Keywords: Acute Kidney Injury - metabolism; Acute Kidney Injury - pathology; Adolescent; Adult; Aged; Case-Control Studies; Child; Endothelium, Vascular - metabolism; Endothelium, Vascular - pathology; Female; Humans; Hypothesis testing; Injuries; Kidney Transplantation - pathology; Kidney Tubules, Distal - blood supply; Kidney Tubules, Distal - metabolism; Kidney Tubules, Distal - pathology; Kidneys; Male; Middle Aged; Neurons; Nitric oxide; Nitric Oxide - metabolism; Nitric Oxide Synthase Type I - metabolism; Nitric Oxide Synthase Type II - metabolism; Nitric Oxide Synthase Type III - metabolism; Reperfusion Injury - metabolism; Reperfusion Injury - pathology; Transplantation, Homologous; Young Adult