APA (7th ed.) Citation

Kwon, O., Hong, S., & Ramesh, G. (2009). Diminished NO generation by injured endothelium and loss of macula densa nNOS may contribute to sustained acute kidney injury after ischemia-reperfusion. American journal of physiology. Renal physiology, 296(1), F25-F33. https://doi.org/10.1152/ajprenal.90531.2008

Chicago Style (17th ed.) Citation

Kwon, Osun, Seok-Min Hong, and Ganesan Ramesh. "Diminished NO Generation by Injured Endothelium and Loss of Macula Densa NNOS May Contribute to Sustained Acute Kidney Injury After Ischemia-reperfusion." American Journal of Physiology. Renal Physiology 296, no. 1 (2009): F25-F33. https://doi.org/10.1152/ajprenal.90531.2008.

MLA (9th ed.) Citation

Kwon, Osun, et al. "Diminished NO Generation by Injured Endothelium and Loss of Macula Densa NNOS May Contribute to Sustained Acute Kidney Injury After Ischemia-reperfusion." American Journal of Physiology. Renal Physiology, vol. 296, no. 1, 2009, pp. F25-F33, https://doi.org/10.1152/ajprenal.90531.2008.