Genetic perturbations suggest a role of the resting potential in regulating the expression of the ion channels of the KCNA and HCN families in octopus cells of the ventral cochlear nucleus - Cao, Xiao-Jie; Oertel, Donata (Hearing research, 2017)
Keywords: Animals; Auditory Pathways - cytology; Auditory Pathways - drug effects; Auditory Pathways - metabolism; Cochlear Nucleus - cytology; Cochlear Nucleus - drug effects; Cochlear Nucleus - metabolism; Gene Expression Regulation; Genetic compensation; Genotype; HCN; Homeostasis; Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels - antagonists & inhibitors; Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels - deficiency; Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels - genetics; KCNA; Kv1.1 Potassium Channel - antagonists & inhibitors; Kv1.1 Potassium Channel - deficiency; Kv1.1 Potassium Channel - genetics; Membrane Potentials - drug effects; Mice, 129 Strain; Mice, Inbred C57BL; Mice, Inbred ICR; Mice, Knockout; Neuronal Plasticity; Patch-Clamp Techniques; Phenotype; Potassium Channel Blockers - pharmacology; Potassium Channels - deficiency; Potassium Channels - genetics; Time Factors; Ventral cochlear nucleus; Voltage-gated ion channels; Homeostasis; KCNA; HCN; Voltage-gated ion channels; Genetic compensation; Ventral cochlear nucleus