Role of A20 in interferon‐α‐mediated functional restoration of myeloid dendritic cells in patients with chronic hepatitis C - Ma, Li; Zhou, Yun; Zhang, Ying; Li, Yuan; Guo, Yonghong; He, Yu; Wang, Jiuping; Lian, Jianqi; Hao, Chunqiu; Moorman, Jonathan P.; Yao, Zhi Q.; Zhou, Yongxing; et al. (Immunology, 2014)
Keywords: A20; Adolescent; Adult; B7-2 Antigen - genetics; B7-2 Antigen - metabolism; Case-Control Studies; chronic infection; Dendritic Cells - drug effects; Dendritic Cells - immunology; Dendritic Cells - metabolism; DNA-Binding Proteins - genetics; DNA-Binding Proteins - metabolism; Female; Gene Expression Regulation - drug effects; Genotype; Hepacivirus - genetics; Hepacivirus - immunology; hepatitis C virus; Hepatitis C, Chronic - drug therapy; Hepatitis C, Chronic - genetics; Hepatitis C, Chronic - immunology; Hepatitis C, Chronic - metabolism; Hepatitis C, Chronic - virology; HLA-DR Antigens - genetics; HLA-DR Antigens - metabolism; Humans; Interferon-alpha - metabolism; Interferon-alpha - pharmacology; Interferon-alpha - therapeutic use; interferon‐α; Interleukin-10 - biosynthesis; Interleukin-12 - biosynthesis; Intracellular Signaling Peptides and Proteins - genetics; Intracellular Signaling Peptides and Proteins - metabolism; Male; Middle Aged; myeloid dendritic cells; Nuclear Proteins - genetics; Nuclear Proteins - metabolism; Original; Receptors, CCR7 - genetics; Receptors, CCR7 - metabolism; RNA, Messenger - genetics; Tumor Necrosis Factor alpha-Induced Protein 3; Viral Load; Young Adult; hepatitis C virus; myeloid dendritic cells; interferon-α; chronic infection; A20