Restoration of Autophagic Flux Improves Endothelial Function in Diabetes Through Lowering Mitochondrial ROS-Mediated eNOS Monomerization - Zhao, Lei; Zhang, Cheng-Lin; He, Lei; Chen, Qinghua; Liu, Limei; Kang, Lijing; Liu, Jian; Luo, Jiang-Yun; Gou, Lingshan; Qu, Dan; Song, Wencong; Lau, Chi Wai; et al. (Diabetes (New York, N.Y.), 2022)
Keywords: Advanced glycosylation end products; Animals; Autophagy; Bioavailability; Chloroquine; Diabetes; Diabetes mellitus; Diabetes Mellitus, Experimental - metabolism; Dimerization; Endothelial cells; Endothelial Cells - metabolism; Endothelium; Endothelium, Vascular - metabolism; Glycosylation; Localization; Mammals - metabolism; Mice; Mice, Inbred C57BL; Mitochondria; Nitric oxide; Nitric Oxide Synthase Type III - genetics; Nitric Oxide Synthase Type III - metabolism; Nitric-oxide synthase; Nutrient deficiency; Oxidative stress; Rapamycin; Reactive oxygen species; Reactive Oxygen Species - metabolism; TOR protein