STAT1 and STAT6 orchestrate Cbs transcription and transsulfur metabolism in microglia and contribute to parkinson’s disease-related neuroinflammation - Liu, Yang; Wang, Zhen; Ma, Ya-Ting; Chen, Yu-Xuan; Qian, Xin-Yi; Hou, Xiao-Ou; Wan, Bo; Ren, Hai-Gang; Hu, Li-Fang (Cellular and molecular life sciences : CMLS, 2025)
Keywords: Antibodies; Biochemistry; Biomedical and Life Sciences; Biomedicine; CBS; Cell Biology; Cystathionine b-synthase; DJ-1; Dopamine receptors; Fibrils; Gene regulation; Genetic factors; Homeostasis; Inflammation; Life Sciences; Lipopolysaccharides; Metabolism; Metabolites; Microglia; Movement disorders; Neurodegenerative diseases; Neurotoxicity; Nitric oxide; Original; Original Article; PARK7 protein; Parkinson's disease; Pathogenesis; Penicillin; Proteins; Signal transduction; STAT1; Stat1 protein; STAT6; Stat6 protein; Stimulation; Sulfur; Synuclein; Transcription activation; Transsulfur metabolism; Tumor necrosis factor-TNF; γ-Interferon; United States--US; Switzerland; China; Shanghai China; STAT6; DJ-1; CBS; STAT1; Transsulfur metabolism; Microglia