FAP Promotes Immunosuppression by Cancer-Associated Fibroblasts in the Tumor Microenvironment via STAT3–CCL2 Signaling - Yang, Xuguang; Lin, Yuli; Shi, Yinghong; Li, Bingji; Liu, Weiren; Yin, Wei; Dang, Yongjun; Chu, Yiwei; Fan, Jia; He, Rui (Cancer research (Chicago, Ill.), 2016)
Keywords: Animals; Cancer-Associated Fibroblasts - physiology; Cell Movement; Chemokine CCL2 - physiology; Female; Focal Adhesion Protein-Tyrosine Kinases - physiology; Gelatinases - physiology; Immune Tolerance; Janus Kinase 2 - physiology; Membrane Proteins - physiology; Mice; Mice, Inbred C57BL; Receptors, Urokinase Plasminogen Activator - physiology; Serine Endopeptidases - physiology; Signal Transduction - physiology; STAT3 Transcription Factor - physiology; Tumor Microenvironment