Proton motive force dissipation precludes interaction of microcin J25 with RNA polymerase, but enhances reactive oxygen species overproduction - Dupuy, Fernando G.; Chirou, María V. Niklison; de Arcuri, Beatriz Fernández; Minahk, Carlos J.; Morero, Roberto D. (Biochimica et biophysica acta, 2009)
Keywords: 2,4 DNP; 2,4-Dinitrophenol - pharmacology; Amino Acid Substitution; Anti-Bacterial Agents - metabolism; Anti-Bacterial Agents - pharmacology; Bacteriocins - genetics; Bacteriocins - metabolism; Bacteriocins - pharmacology; Cell Membrane - drug effects; Cell Membrane - metabolism; DNA-Directed RNA Polymerases - metabolism; E. coli; Escherichia coli - drug effects; Escherichia coli - genetics; Escherichia coli - metabolism; Escherichia coli Proteins - genetics; Escherichia coli Proteins - metabolism; Escherichia coli Proteins - pharmacology; Gene Expression Regulation, Bacterial - drug effects; Microbial Sensitivity Tests; Microcin; Mutation; Oxygen Consumption - drug effects; Peptide uptake; Protein Binding - drug effects; Proton-Motive Force; Reactive oxygen species; Reactive Oxygen Species - metabolism; Uncoupling Agents - pharmacology; 2,4 DNP; Reactive oxygen species; Peptide uptake; Microcin; E. coli