MAP kinase upregulation after hematopoietic differentiation: role of chemotaxis

• Published in: American Journal of Physiology: Cell Physiology Vol. 280; no. 1; pp. C183 - C191
• Main Authors: Lehman, Jason A, Paul, Cassandra C, Baumann, Michael A, Gomez-Cambronero, Julian
• Format: Journal Article
• Language: English
• Published: United States 01.01.2001
• Subjects: Cell Differentiation - drug effects; Cell Differentiation - physiology; Cell Line - cytology; Cell Line - drug effects; Cell Line - metabolism; Chemotaxis - drug effects; Chemotaxis - physiology; Dimethyl Sulfoxide - pharmacology; Granulocyte-Macrophage Colony-Stimulating Factor - metabolism; Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology; Hematopoiesis - drug effects; Hematopoiesis - physiology; HL-60 Cells - cytology; HL-60 Cells - drug effects; HL-60 Cells - metabolism; Humans; Mitogen-Activated Protein Kinase 1 - drug effects; Mitogen-Activated Protein Kinase 1 - metabolism; Mitogen-Activated Protein Kinases - drug effects; Mitogen-Activated Protein Kinases - metabolism; Myeloid Progenitor Cells - cytology; Myeloid Progenitor Cells - drug effects; Myeloid Progenitor Cells - metabolism; Neutrophils - cytology; Neutrophils - drug effects; Neutrophils - metabolism; Phosphorylation - drug effects; Ribosomal Protein S6 Kinases - drug effects; Ribosomal Protein S6 Kinases - metabolism; Tyrosine - metabolism; Up-Regulation - drug effects; Up-Regulation - physiology

Departments of 1  Physiology and Biophysics and 2  Medicine, Wright State University School of Medicine, Dayton 45435; and 3  Research Service, Dayton Veterans Affairs Medical Center, Dayton, Ohio 45428 Mitogen-activated protein kinase (MAPK) isoform p42 is known to be active in exponentially growin...