p53 isoform Δ113p53/Δ133p53 promotes DNA double-strand break repair to protect cell from death and senescence in response to DNA damage - Gong, Lu; Gong, Hongjian; Pan, Xiao; Chang, Changqing; Ou, Zhao; Ye, Shengfan; Yin, Le; Yang, Lina; Tao, Ting; Zhang, Zhenhai; Liu, Cong; Lane, David P; et al. (Cell research, 2015)
Keywords: 631/337/1427/2122; 631/80/82; 692/699/67/1059/485; 692/699/67/581; Animals; Animals, Genetically Modified; Apoptosis - genetics; Biomedical and Life Sciences; Cell Biology; Cell Line; Cellular Senescence - genetics; DNA - genetics; DNA Breaks, Double-Stranded - radiation effects; DNA Ligase ATP; DNA Ligases - biosynthesis; DNA Repair - genetics; DNA-Binding Proteins - biosynthesis; DNA-Binding Proteins - metabolism; G2 Phase Cell Cycle Checkpoints - genetics; G2 Phase Cell Cycle Checkpoints - radiation effects; Humans; Life Sciences; Original; original-article; Promoter Regions, Genetic - genetics; Protein Binding; Protein Isoforms - genetics; Rad51 Recombinase - biosynthesis; RNA Interference; RNA, Small Interfering; Tumor Suppressor Protein p53 - genetics; Zebrafish; Zebrafish Proteins - biosynthesis; Zebrafish Proteins - genetics; SSA; senescence; Δ113p53/Δ133p53; cell death; p53 isoform; HR; NHEJ