Activation of invariant natural killer T cells impedes liver regeneration by way of both IFN‐γ‐ and IL‐4‐dependent mechanisms - Yin, Shi; Wang, Hua; Bertola, Adeline; Feng, Dechun; Xu, Ming‐jiang; Wang, Yan; Gao, Bin (Hepatology (Baltimore, Md.), 2014)
Keywords: Animals; Antigens, CD1d - genetics; Antigens, CD1d - physiology; Cell Proliferation - drug effects; Galactosylceramides - pharmacology; Hepatectomy; Hepatocytes - drug effects; Hepatocytes - pathology; In Vitro Techniques; Interferon-gamma - deficiency; Interferon-gamma - genetics; Interferon-gamma - physiology; Interleukin-4 - deficiency; Interleukin-4 - genetics; Interleukin-4 - physiology; Liver - drug effects; Liver - physiology; Liver - surgery; Liver Regeneration - drug effects; Liver Regeneration - physiology; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Models, Animal; Natural Killer T-Cells - pathology; Natural Killer T-Cells - physiology; STAT6 Transcription Factor - deficiency; STAT6 Transcription Factor - genetics; STAT6 Transcription Factor - physiology