Overexpression of semicarbazide sensitive amine oxidase in the cerebral blood vessels in patients with Alzheimer’s disease and cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy - Ferrer, I.; Lizcano, J.M.; Hernández, M.; Unzeta, M. (Neuroscience letters, 2002)
Keywords: Aged; Aged, 80 and over; Alzheimer disease; Alzheimer Disease - enzymology; Alzheimer Disease - physiopathology; Amine Oxidase (Copper-Containing) - metabolism; Amyloid beta-Peptides - metabolism; Biological and medical sciences; Blood Vessels - enzymology; Blood Vessels - pathology; Blood Vessels - physiopathology; Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy; Cerebral blood vessels; Cerebral Cortex - blood supply; Cerebral Cortex - enzymology; Cerebral Cortex - physiopathology; Cu/Zn superoxide dismutase 1; Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases; Dementia, Multi-Infarct - enzymology; Dementia, Multi-Infarct - physiopathology; Female; Humans; Immunohistochemistry; Male; Medical sciences; Neurology; Oxidative stress; Oxidative Stress - physiology; Semicarbazide sensitive amine oxidase; Up-Regulation - physiology; βA4 Amyloid; Oxidative stress; Semicarbazide sensitive amine oxidase; Alzheimer disease; Cerebral blood vessels; Cu/Zn superoxide dismutase 1; Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy; βA4 Amyloid; Human; Nervous system diseases; Enzyme; Central nervous system; Gene overexpression; Cerebral disorder; β Amyloid protein; Blood vessel; Central nervous system disease; CADASIL syndrome; Degenerative disease; Circulatory system; Brain (vertebrata)