Cortical blindness in acute carbon monoxide poisoning

A 3-year-old boy had persistent cortical blindness following acute carbon monoxide poisoning. He was believed to have suffered anoxic brain damage due to incomplete combustion of the briquette-type solid fuel Computed tomographic (CT) scan of the brain and visual evoked potentials (VEP) in the early...

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Published inBrain & development (Tokyo. 1979) Vol. 7; no. 5; pp. 516 - 519
Main Authors Katafuchi, Yukihiko, Nishimi, Toshihiro, Yamaguchi, Yoichiro, Matsuishi, Toyojiro, Kimura, Yoshiyuki, Otaki, Etsuo, Yamashita, Yushiro
Format Journal Article
LanguageEnglish
Published Amsterdam Elsevier B.V 1985
Elsevier Science
Subjects
Acute carbon monoxide poisoning
Biological and medical sciences
Blindness - etiology
Carbon Monoxide Poisoning - complications
Chemical and industrial products toxicology. Toxic occupational diseases
Child, Preschool
cortical blindness
Encephalomalacia - diagnostic imaging
Encephalomalacia - etiology
Encephalomalacia - physiopathology
Evoked Potentials, Visual
Gas, fumes
Humans
leukomalacia
Male
Medical sciences
Tomography, X-Ray Computed
Toxicology
visual evoked potentials
Acute carbon monoxide poisoning
visual evoked potentials
leukomalacia
cortical blindness
Nervous system diseases
Poisoning
Cortical blindness
Carbon monoxide
Toxicity
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Abstract A 3-year-old boy had persistent cortical blindness following acute carbon monoxide poisoning. He was believed to have suffered anoxic brain damage due to incomplete combustion of the briquette-type solid fuel Computed tomographic (CT) scan of the brain and visual evoked potentials (VEP) in the early stage were normal. However, on the 20th hospital day CT scan showed leukomalacia and VEP showed an absence of N1-, and P1-waves which was well correlated with the clinical feature at that time.
AbstractList A 3-year-old boy had persistent cortical blindness following acute carbon monoxide poisoning. He was believed to have suffered anoxic brain damage due to incomplete combustion of the briquette-type solid fuel. Computed tomographic (CT) scan of the brain and visual evoked potentials (VEP) in the early stage were normal. However, on the 20th hospital day CT scan showed leukomalacia and VEP showed an absence of N1-, and P1-waves which was well correlated with the clinical feature at that time.
Author Yamaguchi, Yoichiro
Kimura, Yoshiyuki
Nishimi, Toshihiro
Matsuishi, Toyojiro
Otaki, Etsuo
Yamashita, Yushiro
Katafuchi, Yukihiko
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Cites_doi 10.1136/bmj.1.5849.318
10.1016/S0079-6123(08)60181-8
10.1097/00005053-194011000-00008
10.1001/archneur.1974.00490330017003
10.1016/S0140-6736(83)91705-1
10.1001/archneur.1979.00500370068016
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Issue 5
Keywords Acute carbon monoxide poisoning
visual evoked potentials
leukomalacia
cortical blindness
Nervous system diseases
Poisoning
Cortical blindness
Carbon monoxide
Toxicity
Language English
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Snippet A 3-year-old boy had persistent cortical blindness following acute carbon monoxide poisoning. He was believed to have suffered anoxic brain damage due to...
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SubjectTerms Acute carbon monoxide poisoning
Biological and medical sciences
Blindness - etiology
Carbon Monoxide Poisoning - complications
Chemical and industrial products toxicology. Toxic occupational diseases
Child, Preschool
cortical blindness
Encephalomalacia - diagnostic imaging
Encephalomalacia - etiology
Encephalomalacia - physiopathology
Evoked Potentials, Visual
Gas, fumes
Humans
leukomalacia
Male
Medical sciences
Tomography, X-Ray Computed
Toxicology
visual evoked potentials
Title Cortical blindness in acute carbon monoxide poisoning
URI https://dx.doi.org/10.1016/S0387-7604(85)80121-2
https://www.ncbi.nlm.nih.gov/pubmed/4083389
Volume 7
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