PFKFB3 facilitates cell proliferation and migration in anaplastic thyroid carcinoma via the WNT/β‐catenin signaling pathway - Deng, Jinmei; Cheng, Yanglei; Li, Hai; He, Xiaoying; Yu, Shuang; Ma, Jiajing; Li, Xuhui; Chen, Jie; Xiao, Haipeng; Guan, Hongyu; Li, Yanbing (Endocrine, 2024)
Keywords: AKT protein; Animals; Cell growth; Cell Line, Tumor; Cell migration; Cell Movement; Cell Proliferation; Cyclin D1; Diabetes; Down-regulation; Endocrinology; Gelatinase B; Gene expression; Glycolysis; Humanities and Social Sciences; Humans; Immunohistochemistry; Internal Medicine; Kinases; Lactic acid; Medicine; Medicine & Public Health; Metalloproteinase; Metastases; Mice; Mice, Nude; Molecular modelling; multidisciplinary; Nuclear transport; Original Article; Phosphofructokinase-2 - genetics; Phosphofructokinase-2 - metabolism; Science; Signal transduction; Thyroid cancer; Thyroid carcinoma; Thyroid Carcinoma, Anaplastic - genetics; Thyroid Carcinoma, Anaplastic - metabolism; Thyroid Neoplasms - genetics; Thyroid Neoplasms - metabolism; Tumors; Western blotting; Wnt protein; Wnt Signaling Pathway; β-Catenin; PFKFB3; Proliferation; WNT/β‐catenin; Migration; Anaplastic thyroid carcinoma (ATC)