Gisenoside Rg1 attenuates cadmium-induced neurotoxicity in vitro and in vivo by attenuating oxidative stress and inflammation - Ren, Teng-Teng; Yang, Jia-Ying; Wang, Jun; Fan, Sheng-Rui; Lan, Rongfeng; Qin, Xiao-Yan (Inflammation research, 2021)
Keywords: AKT protein; Allergology; Animal behavior; Animals; Anti-Inflammatory Agents - pharmacology; Anti-Inflammatory Agents - therapeutic use; Antioxidants; Apoptosis - drug effects; Astrocytes; Biocompatibility; Biomedical and Life Sciences; Biomedicine; Brain; Brain - drug effects; Brain - immunology; Brain - pathology; Brain-derived neurotrophic factor; Cadmium; Cell Survival - drug effects; Cells, Cultured; Cognitive ability; Cytokines - genetics; Cytokines - immunology; Dermatology; Ginseng; Ginsenosides - pharmacology; Ginsenosides - therapeutic use; Glial fibrillary acidic protein; IL-1β; Immunology; Inflammation; Interleukin 6; Intestine; Intestine, Small - drug effects; Intestine, Small - pathology; Kidney - drug effects; Kidney - pathology; Kidneys; Lipid peroxidation; Lipids; Liver - drug effects; Liver - pathology; Male; Mice; Mice, Inbred C57BL; Microglia; Microglia - drug effects; Microglia - pathology; Neurology; Neurons - drug effects; Neurons - pathology; Neuroprotective agents; Neuroprotective Agents - pharmacology; Neuroprotective Agents - therapeutic use; Neurotoxicity; Neurotoxicity Syndromes - drug therapy; Neurotoxicity Syndromes - immunology; Neurotoxicity Syndromes - pathology; Original Research Article; Oxidative stress; Oxidative Stress - drug effects; Oxidoreductases - immunology; Peroxidation; Pharmacology/Toxicology; Rheumatology; Small intestine; Testis - drug effects; Testis - pathology; Toxicity; TrkB receptors; Tumor necrosis factor-α; IBA1; TNF-α; Rg1; HES-1; Cd neurotoxicity; GFAP