Porphyromonas gingivalis Promotes Colorectal Carcinoma by Activating the Hematopoietic NLRP3 Inflammasome - Wang, Xi; Jia, Yiqun; Wen, Liling; Mu, Wenxin; Wu, Xianrui; Liu, Tao; Liu, Xiangqi; Fang, Juan; Luan, Yizhao; Chen, Ping; Gao, Jinlong; Nguyen, Ky-Anh; et al. (Cancer research (Chicago, Ill.), 2021)
Keywords: Animals; Apoptosis; Bacteroidaceae Infections - complications; Bacteroidaceae Infections - immunology; Bacteroidaceae Infections - microbiology; Bacteroidaceae Infections - pathology; Carcinogenesis - immunology; Carcinogenesis - metabolism; Carcinogenesis - pathology; Cell Proliferation; Colorectal Neoplasms - immunology; Colorectal Neoplasms - metabolism; Colorectal Neoplasms - microbiology; Colorectal Neoplasms - pathology; Gene Expression Regulation, Neoplastic; Humans; Inflammasomes - immunology; Inflammasomes - metabolism; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, Nude; Myeloid Cells - immunology; Myeloid Cells - metabolism; Myeloid Cells - microbiology; Myeloid Cells - pathology; Neoplastic Stem Cells - immunology; Neoplastic Stem Cells - metabolism; Neoplastic Stem Cells - microbiology; Neoplastic Stem Cells - pathology; NLR Family, Pyrin Domain-Containing 3 Protein - physiology; Porphyromonas gingivalis - pathogenicity; Prognosis; Survival Rate; Tumor Cells, Cultured; Tumor Microenvironment - immunology; Xenograft Model Antitumor Assays