Inhibition of extracellular signal‐regulated kinases 1/2 provides neuroprotection in spinal cord ischemia/reperfusion injury in rats: relationship with the nuclear factor‐κB‐regulated anti‐apoptotic mechanisms - Lu, Kang; Liang, Cheng‐Loong; Liliang, Po‐Chou; Yang, Chih‐Hui; Cho, Chung‐Lung; Weng, Hui‐Ching; Tsai, Yu‐Duan; Wang, Kuo‐Wei; Chen, Han‐Jung (Journal of neurochemistry, 2010)
Keywords: apoptosis; Biological and medical sciences; Cardiology. Vascular system; cellular inhibitor of apoptosis protein 2; Cerebrospinal fluid. Meninges. Spinal cord; extracellular signal‐regulated kinases 1/2; ischemia‐reperfusion injury; Medical sciences; Nervous system (semeiology, syndromes); Neurology; nuclear factor‐κB; spinal cord; Phosphorylation; cellular inhibitor of apoptosis protein 2; Extracellular signal-regulated protein kinase; Rat; Central nervous system; Cardiovascular disease; Vascular disease; Signal transduction; Reperfusion; Spinal cord trauma; Transcription factor NFκB; spinal cord; Nervous system diseases; Enzyme; Transferases; Rodentia; Caspase; Mitogen-activated protein kinase; Survival; Protein; extracellular signal-regulated kinases 1/2; Spinal cord ischemia; ischemia-reperfusion injury; nuclear factor-κB; Peptidases; Vertebrata; Mammalia; Treatment; Cell death; Central nervous system disease; Hydrolases; Spinal cord disease; Apoptosis