Glucose intolerance but normal satiety in mice with a null mutation in the glucagon–like peptide 1 receptor gene - Scrocchi, L.A.; Brown, T.J.; Maclusky, N.; Brubaker, P.L.; Auerbach, A.B.; Joyner, A.L.; Drucker, D.J. (Nature medicine, 1996)
Keywords: Animals; Biomedical and Life Sciences; Biomedicine; Blood Glucose - analysis; Cancer Research; Female; Gene Deletion; Glucagon - pharmacology; Glucagon-Like Peptide 1; Glucagon-Like Peptide-1 Receptor; Glucose - metabolism; Glucose - pharmacology; Glucose Intolerance; Infectious Diseases; Insulin - blood; Male; Metabolic Diseases; Mice; Molecular Medicine; Neurosciences; Peptide Fragments - pharmacology; Protein Precursors - pharmacology; Rats; Receptors, Glucagon - genetics; Receptors, Glucagon - metabolism