Helicobacter pylori delays neutrophil apoptosis but also drives the formation of cells with a leaky plasma membrane: Implications for inflammation - Thai, Tran Duong; Kamsom, Chatcharin; Phoksawat, Wisitsak; Nithichanon, Arnone; Faksri, Kiatichai; Sripa, Banchob; Edwards, Steven W.; Salao, Kanin (Molecular immunology, 2025)
Keywords: Antigens, Bacterial - immunology; Apoptosis; Apoptosis - immunology; Bacterial Proteins - immunology; basins; cagA; Cell Membrane - immunology; Cell Membrane - metabolism; Cell Membrane - microbiology; Cell Membrane - pathology; cell viability; coculture; Coculture Techniques; Helicobacter Infections - immunology; Helicobacter Infections - pathology; Helicobacter pylori; Helicobacter pylori - immunology; Humans; inflammation; Inflammation - immunology; Inflammation - microbiology; Inflammation - pathology; longevity; mixed infection; Neutrophil Activation - immunology; Neutrophils; Neutrophils - immunology; Neutrophils - microbiology; Neutrophils - pathology; Opisthorchis viverrini; pathogenicity; phagocytosis; Phagocytosis - immunology; plasma membrane; propidium; public health; reactive oxygen species; Reactive Oxygen Species - metabolism; Helicobacter pylori; Opisthorchis viverrini; cagA; Neutrophils; Apoptosis