RETRACTED: Low-expressed GAS5 injure myocardial cells and progression of chronic heart failure via regulation of miR-223-3P
This article has been retracted: please see Elsevier Policy on Article Withdrawal (https://www.elsevier.com/about/our-business/policies/article-withdrawal). This article has been retracted at the request of the Editor-in-Chief. After a thorough investigation, the Editor has concluded that the accept...
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| Published in | Experimental and molecular pathology Vol. 117; p. 104529 |
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| Main Authors | , , , , , , |
| Format | Journal Article |
| Language | English |
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01.12.2020
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| Abstract | This article has been retracted: please see Elsevier Policy on Article Withdrawal (https://www.elsevier.com/about/our-business/policies/article-withdrawal).
This article has been retracted at the request of the Editor-in-Chief.
After a thorough investigation, the Editor has concluded that the acceptance of this article was partly based upon the positive advice of one illegitimate reviewer report. The report was submitted from an email account which was provided to the journal as a suggested reviewer during the submission of the article. Although purportedly a real reviewer account, the Editor has concluded that this was not of an appropriate, independent reviewer.
This manipulation of the peer-review process represents a clear violation of the fundamentals of peer review, our publishing policies, and publishing ethics standards. Apologies are offered to the reviewer whose identity was assumed and to the readers of the journal that this deception was not detected during the submission process. |
|---|---|
| AbstractList | This article has been retracted: please see Elsevier Policy on Article Withdrawal (https://www.elsevier.com/about/our-business/policies/article-withdrawal).
This article has been retracted at the request of the Editor-in-Chief.
After a thorough investigation, the Editor has concluded that the acceptance of this article was partly based upon the positive advice of one illegitimate reviewer report. The report was submitted from an email account which was provided to the journal as a suggested reviewer during the submission of the article. Although purportedly a real reviewer account, the Editor has concluded that this was not of an appropriate, independent reviewer.
This manipulation of the peer-review process represents a clear violation of the fundamentals of peer review, our publishing policies, and publishing ethics standards. Apologies are offered to the reviewer whose identity was assumed and to the readers of the journal that this deception was not detected during the submission process. Chronic heart failure (CHF) is a common disease in clinical practice, and its incidence has been increasing in recent years. Understanding the pathogenesis of CHF is the key to its future clinical diagnosis and treatment. Molecular research is a hot topic in modern hospitals, and long non-coding RNA (LncRNA) has been gradually understood and applied in many diseases. The situation of LncRNA GAS5 in CHF is still unclear, so this experiment will investigate the situation of GAS5 in CHF and its effect on myocardial cells, aiming to gain a preliminary understanding of the mechanism of GAS5's effect on CHF. In this study, the expression of GAS5 and miR-223-3p in peripheral blood of CHF patients and healthy subjects was first detected, GAS5 was low in CHF while miR-223-3p was high (P < 0.05). Subsequently, ROC curve analysis showed that GAS5 and miR-223-3p had good predictive value for the occurrence and recurrence of CHF. Secondly, through in vitro experiments, we found that inhibition of GAS5 with elevated expression of miR-223-3p decreased the proliferative capacity of cardiomyocytes and increased apoptotic capacity and inflammatory factors (P < 0.050). Through dual luciferase reporter and RNA immunoprecipitation experiment, we found that miR-223-3p was regulated by GAS5 in a targeted manner. Chronic heart failure (CHF) is a common disease in clinical practice, and its incidence has been increasing in recent years. Understanding the pathogenesis of CHF is the key to its future clinical diagnosis and treatment. Molecular research is a hot topic in modern hospitals, and long non-coding RNA (LncRNA) has been gradually understood and applied in many diseases. The situation of LncRNA GAS5 in CHF is still unclear, so this experiment will investigate the situation of GAS5 in CHF and its effect on myocardial cells, aiming to gain a preliminary understanding of the mechanism of GAS5's effect on CHF. In this study, the expression of GAS5 and miR-223-3p in peripheral blood of CHF patients and healthy subjects was first detected, GAS5 was low in CHF while miR-223-3p was high (P < 0.05). Subsequently, ROC curve analysis showed that GAS5 and miR-223-3p had good predictive value for the occurrence and recurrence of CHF. Secondly, through in vitro experiments, we found that inhibition of GAS5 with elevated expression of miR-223-3p decreased the proliferative capacity of cardiomyocytes and increased apoptotic capacity and inflammatory factors (P < 0.050). Through dual luciferase reporter and RNA immunoprecipitation experiment, we found that miR-223-3p was regulated by GAS5 in a targeted manner.Chronic heart failure (CHF) is a common disease in clinical practice, and its incidence has been increasing in recent years. Understanding the pathogenesis of CHF is the key to its future clinical diagnosis and treatment. Molecular research is a hot topic in modern hospitals, and long non-coding RNA (LncRNA) has been gradually understood and applied in many diseases. The situation of LncRNA GAS5 in CHF is still unclear, so this experiment will investigate the situation of GAS5 in CHF and its effect on myocardial cells, aiming to gain a preliminary understanding of the mechanism of GAS5's effect on CHF. In this study, the expression of GAS5 and miR-223-3p in peripheral blood of CHF patients and healthy subjects was first detected, GAS5 was low in CHF while miR-223-3p was high (P < 0.05). Subsequently, ROC curve analysis showed that GAS5 and miR-223-3p had good predictive value for the occurrence and recurrence of CHF. Secondly, through in vitro experiments, we found that inhibition of GAS5 with elevated expression of miR-223-3p decreased the proliferative capacity of cardiomyocytes and increased apoptotic capacity and inflammatory factors (P < 0.050). Through dual luciferase reporter and RNA immunoprecipitation experiment, we found that miR-223-3p was regulated by GAS5 in a targeted manner. |
| ArticleNumber | 104529 |
| Author | Li, Bo Li, Gang Qiang, Xu Du, Pang Jin, Dayong Liu, Haochen Guo, Jianjun |
| Author_xml | – sequence: 1 givenname: Gang surname: Li fullname: Li, Gang organization: Department of Cardiovascular Surgery, Xian Children's Hospital, No. 69 Xijuyuan Land, Xian, 710003, Shannxi Province, China – sequence: 2 givenname: Pang surname: Du fullname: Du, Pang organization: Department of Radiology, Tangdu Hospital, Xian, 710003, Shannxi, China – sequence: 3 givenname: Xu surname: Qiang fullname: Qiang, Xu organization: Department of Cardiovascular Surgery, Xian Children's Hospital, No. 69 Xijuyuan Land, Xian, 710003, Shannxi Province, China – sequence: 4 givenname: Dayong surname: Jin fullname: Jin, Dayong organization: Department of Radiology, Tangdu Hospital, Xian, 710003, Shannxi, China – sequence: 5 givenname: Haochen surname: Liu fullname: Liu, Haochen organization: Department of Cardiovascular Surgery, Xian Children's Hospital, No. 69 Xijuyuan Land, Xian, 710003, Shannxi Province, China – sequence: 6 givenname: Bo surname: Li fullname: Li, Bo organization: Department of Cardiovasology, Tangdu Hospital, Xian, 710003, Shannxi, China – sequence: 7 givenname: Jianjun surname: Guo fullname: Guo, Jianjun email: jianjunguo452@163.com organization: Department of Cardiovascular Surgery, Xian Children's Hospital, No. 69 Xijuyuan Land, Xian, 710003, Shannxi Province, China |
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| Cites_doi | 10.1002/ejhf.1146 10.1002/ejhf.839 10.1007/s12350-015-0336-8 10.1002/ejhf.669 10.1016/j.ccell.2018.03.006 10.1016/j.canlet.2018.06.039 10.1007/s12035-016-0246-z 10.1093/eurheartj/ehx460 10.1016/j.bbi.2018.02.013 10.14715/cmb/2017.63.4.1 10.3892/mmr.2017.7653 10.15420/cfr.2016:25:2 10.3389/fphar.2017.00464 10.1177/2048872617748553 10.1016/j.tox.2017.05.007 10.1002/jcb.28789 10.18632/oncotarget.19197 10.1161/CIRCULATIONAHA.116.025941 10.1016/S0140-6736(17)32520-5 10.1161/HYPERTENSIONAHA.116.07259 |
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| Snippet | This article has been retracted: please see Elsevier Policy on Article Withdrawal (https://www.elsevier.com/about/our-business/policies/article-withdrawal).... Chronic heart failure (CHF) is a common disease in clinical practice, and its incidence has been increasing in recent years. Understanding the pathogenesis of... |
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| SubjectTerms | Adult Aged Apoptosis - genetics Cell Line Female Heart Failure - blood Heart Failure - pathology Humans Interleukin-6 - blood Male MicroRNAs - blood Middle Aged Myocardium - metabolism Myocardium - pathology RNA, Long Noncoding - blood Tumor Necrosis Factor-alpha - blood |
| Title | RETRACTED: Low-expressed GAS5 injure myocardial cells and progression of chronic heart failure via regulation of miR-223-3P |
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