Gut dysbiosis induces the development of pre-eclampsia through bacterial translocation - Chen, Xia; Li, Pan; Liu, Mian; Zheng, Huimin; He, Yan; Chen, Mu-Xuan; Tang, Wenli; Yue, Xiaojing; Huang, Yongxin; Zhuang, Lingling; Wang, Zhijian; Zhong, Mei; et al. (Gut, 2020)
Keywords: Animals; Antibiotics; Antigens; Bacteria; Bacterial Translocation; Blood Pressure; Case-Control Studies; CD4 Lymphocyte Count; Chemokines - genetics; Creatinine; Creatinine - blood; Cytokines - genetics; Digestive system; Discriminant analysis; Disease; Disease Models, Animal; Dysbacteriosis; Dysbiosis - complications; Dysbiosis - physiopathology; Embryos; Faecalibacterium; Feces - microbiology; Female; Fetal Resorption - microbiology; Fetuses; Flow cytometry; Fusobacteria; Fusobacterium; Gastrointestinal Microbiome; Gastrointestinal tract; Gene expression; Gestation; Gut microbiota; Gynecology; Hospitals; Humans; Hybridization; Hypertension; Immune response; Immunoglobulins; immunology; Inflammation; Insulin resistance; intestinal bacteria; intestinal barrier function; Intestinal microflora; Intestine, Small - immunology; Metabolic disorders; Metabolic syndrome; Mice; Microbiomes; Microbiota; Obstetrics; Opportunist infection; Pathogenesis; Placenta; Placenta - immunology; Placenta - metabolism; Placenta - microbiology; Pre-eclampsia; Pre-Eclampsia - microbiology; Pre-Eclampsia - physiopathology; Preeclampsia; Pregnancy; Probiotics; Proteins; Proteinuria; Proteinuria - urine; RNA, Messenger - metabolism; rRNA 16S; Small intestine; Spleen; Statistical analysis; Studies; T-Lymphocytes, Regulatory; Taxonomy; Th17 Cells; Transplantation; Urine; Veillonella; United States--US; China; California; bacterial translocation; intestinal bacteria; intestinal barrier function; immunology