APA (7th ed.) Citation

Qin, Y., Cai, M., Jin, H., Huang, W., Zhu, C., Bozec, A., . . . Chen, Z. (2022). Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation of fibroblast-like synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 pathways. Annals of the rheumatic diseases, 81(11), 1504-1514. https://doi.org/10.1136/ard-2022-222605

Chicago Style (17th ed.) Citation

Qin, Yi, Ming-Long Cai, Hui-Zhi Jin, Wei Huang, Chen Zhu, Aline Bozec, Jingang Huang, and Zhu Chen. "Age-associated B Cells Contribute to the Pathogenesis of Rheumatoid Arthritis by Inducing Activation of Fibroblast-like Synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 Pathways." Annals of the Rheumatic Diseases 81, no. 11 (2022): 1504-1514. https://doi.org/10.1136/ard-2022-222605.

MLA (9th ed.) Citation

Qin, Yi, et al. "Age-associated B Cells Contribute to the Pathogenesis of Rheumatoid Arthritis by Inducing Activation of Fibroblast-like Synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 Pathways." Annals of the Rheumatic Diseases, vol. 81, no. 11, 2022, pp. 1504-1514, https://doi.org/10.1136/ard-2022-222605.